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The NAMS 2014 recommendations guide healthcare providers on treating health issues in midlife women, emphasizing individualized care and informed decision-making.

Scientists made a mouse that can be made to lose hair and then grow it back.

LGD1069 effectively prevents breast tumors in mice without toxicity.

Fluorescent proteins in mouse models effectively visualize tumor blood vessel growth.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

Increased ornithine decarboxylase makes normally tumor-resistant mice more sensitive to tumors.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Researchers created a mouse model for Cushing's syndrome to study glucocorticoid excess and potential treatments.

Researchers developed a mouse model that tracks hair growth using bioluminescence, improving accuracy in studying hair cycles.

Overexpressing NSD3 in mice causes breast cancer-like tumors and gland abnormalities.

A mouse model was created to study hair loss similar to humans.

A new mouse model helps study melanocyte cells using GFP expression.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

Gender-affirming hormone therapy affects metabolism differently based on treatment type.

The study concludes that a genetic mutation in TFM mice leads to reduced androgen receptor activity, affecting the body's response to male hormones.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

Estrogen is important for keeping adult mouse nipple skin healthy by controlling certain cell signals.

Increasing COX-2 in mouse skin causes bigger sebaceous glands and thinner hair, but stopping COX-2 can reverse hair thinning.

Ovariectomized mice mimic postmenopausal hair loss, and estradiol helps maintain hair density.

The mouse models are effective for testing new hair loss treatments.

A testosterone-induced hair loss model in mice was successfully created for future research and treatment testing.

Androgen receptors are active in many tissues of both male and female mice, not just reproductive organs.

The model using human skin on mice helps study human sebaceous glands.

Disrupting the Flcn gene in mice causes early kidney cysts and tumors, which can be treated with rapamycin.

Researchers created a new mouse model for studying scleroderma.

The transgene likely activated an oncogene or interrupted a tumor suppressor gene, causing melanoma in mice.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.