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Researchers developed a mouse model that tracks hair growth using bioluminescence, improving accuracy in studying hair cycles.

Tsc2-deficient stem cells can help understand and treat TSC-related tumors.

Introducing the OTC gene improved symptoms in mice with OTC deficiency.

Disrupting the Tsc2 gene in certain cells leads to thicker skin, larger hair, and changes in hair growth signaling, which can be partly reversed with specific treatment.

Introducing the rat OTC gene partially corrected OTC deficiency in mice.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

The Trichodysplasia spinulosa virus protein can cause abnormal hair growth in mice.

Transferrin receptor 1 is essential for normal function in the intestines, pancreas, and skin.

Tgm2 helps stabilize dying cells and aids fibroblast attachment to the extracellular matrix.

TSC2-/meth cells can cause skin lesions, hair growth, and lung issues, and may be treated with chromatin remodeling agents.

A new mouse model for vitiligo helps study immune responses and potential treatments.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

New mouse models help study melanocytic cells for melanoma research.

Sox13 is a marker for early hair follicle development but not essential for skin and hair growth.

Deleting the Trf1 protein in mice is safe and may help prevent cancer without major side effects.

FoxN1 overexpression in young mice harms immune cell and skin development.

The DiLiCre mouse model is an effective tool for precise genome editing using light.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Lack of functional CYLD in mice leads to early aging and cancer.

Activated β-catenin affects hair growth and skin thickness, and changes are reversible.

Thymus-derived Tregs, not peripherally-derived Tregs, primarily regulate type 1 diabetes in the NOD mouse model.

Tabby mutations in mice affect hair follicle development and help study genetic mapping and certain medical conditions.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

Melanoma cells lose their ability to form tumors when placed in a zebrafish embryo environment.

CD8+ T cells play a key role in graft-versus-host disease in certain mice models.

TNF alpha in skin cells causes weight loss, hair and fat issues, and skin inflammation in mice.

Overexpressing GLI-1 in mice skin can cause tumors like human basal cell carcinomas.