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Blue-light activation of TrkA improves hair-follicle stem cells' ability to become neurons and glial cells.

Telocytes, cells with long extensions, are vital for hair growth because they produce Wnt signals, which are necessary for hair follicle regeneration.

Key genes and pathways control sheep hair growth phases.

Keloid scars may form due to changes in skin cell characteristics and specific protein signaling.

ILC1-like cells can cause alopecia areata by affecting hair follicles.

Ovarian hyperthecosis can cause high testosterone and symptoms like hair loss, and surgery can help improve these symptoms.

A mutation in mice causes hair loss and skin issues due to a defect in a gene affecting cell adhesion.

Antizyme slows skin tumor growth by reducing cell growth in mice.

Increased ODC activity leads to skin tumors by recruiting stem cells, not by toxic byproducts.

Hair loss from telogen effluvium happens when a disturbance or imbalance causes hair to fall out prematurely.

Activated erbB-2 in mice skin causes severe skin and hair abnormalities.

Altering the keratin 17 gene in mice hair follicles caused temporary hair issues, but changes were minimal and short-lived.

Ovol2 is crucial for hair growth and skin healing by controlling cell movement and growth.

QLT0267 stops hair follicle cell growth and movement.

Latanoprost eye drops caused excessive cheek hair growth and eyelash whitening in a woman.

Blocking YAP/TAZ could be a new way to treat skin cancer.

Stimulating OR10J1 can promote hair growth and may help treat hair loss.

Increased 14-3-3 proteins may block hair cycle regression, causing hair loss.

A patient's hair loss after a browlift surgery improved on its own over 8 months.

A new method can detect mutations in mice by observing changes in hair follicle cells.

Epidermal growth factor causes hair loss by triggering cell death and changing the hair cycle.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

A targetoid hair regrowth pattern indicates recovery in alopecia areata.

Removing both Atr and Trp53 genes in adult mice causes severe tissue damage and death due to DNA damage.

Telocytes in the scalp may help with skin regeneration and maintenance.

Loss of TET2 increases the risk of skin and oral cancer.

The malignant pilomatricoma showed strong epithelial keratin expression, suggesting it may not calcify.

Blocking specific proteins can help remove aging cells and might treat age-related diseases and promote hair growth.