Search

everythinglearnresearchcommunity

for

Search:↓

Androgenetic alopecia is a common hair loss condition caused by testosterone effects on hair follicles, leading to thinner, shorter, and less pigmented hair, diagnosed using scalp dermoscopy and treated with topical minoxidil, antiandrogen agents, and 5-alpha reductase inhibitors.

An extract from Quercus acutissima bark was found to reduce sebum production and block an enzyme linked to acne.

Male androgenetic alopecia involves hair follicle miniaturization due to DHT, with potential treatments using inhibitors and blockers.

The research gives new understanding on how human steroid 5α-reductases work and how drugs like finasteride inhibit them, which could help in creating new drugs.

The equine epididymis mainly uses type 1 5α-reductase, and both finasteride and dutasteride can inhibit its activity.

Inhibitors of the enzyme 5 alpha-reductase could potentially treat disorders like prostate cancer and baldness.

Testosterone needs to be converted to DHT to reduce stress response in male rats.

Ginseng, especially red ginseng, may help regrow hair and block a hair loss-related enzyme.

New research is needed to create better drugs that block the enzyme responsible for conditions like male baldness and prostate enlargement.

Testosterone increases acidification in the vas deferens by up-regulating V-ATPase.

Early surgery reduces seizure risk in drug-resistant epilepsy; testosterone to DHT conversion doesn't affect muscle anabolism; veterans with PTSD likely to have riskier opioid use; bevacizumab helps in hereditary bleeding disorder.

Combining D-004 and finasteride is more effective at reducing prostate enlargement than using either alone.

Testosterone and dihydrotestosterone slow hair cell growth, while estradiol has no effect.

Testosterone can slow down hair growth when combined with certain cells from bald scalps, and this effect can be blocked by an androgen receptor blocker.

The new compounds moderately block a specific enzyme and strongly counteract a male hormone, suggesting potential for treating certain male-related health conditions.

Citrullus colocynthis fruit extract and its compound may help treat enlarged prostate.

Testosterone reduces heart damage and inflammation after injury.

5α-reductase inhibitors, like Finasteride and Dutasteride, help manage benign prostatic hyperplasia.

Heart-specific steroid metabolism is crucial in cardiac hypertrophy.

Testosterone boosts red blood cell production and changes iron metabolism without needing dihydrotestosterone.

High testosterone to epitestosterone ratio in hair could predict male-pattern baldness.

Cinnamomi cortex may help treat prostate enlargement by reducing related proteins.

Dihydrotestosterone can be made from dehydroepiandrosterone in skin cells without needing testosterone.

White wax and policosanol from white wax effectively reduced hair loss and promoted hair growth in mice better than a known hair growth drug.

Testosterone protects female heart cells from stress by activating estrogen receptors.

Testosterone may have a dual role in COVID-19, potentially worsening outcomes in men, and testosterone therapy could help some patients, but more research is needed.

The method effectively measures drug impact on DHT levels in prostate treatments.

Testosterone boosts fluid and electrolyte secretion in seminal vesicles, aiding sperm function.

Formalin-induced pain increases testosterone breakdown in the rat brain and spinal cord.

Prostaglandin D2 increases testosterone production in skin cells through a process involving reactive oxygen species, and antioxidants may help treat hair loss.