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RANKL improves the immune response against herpes simplex virus by enhancing T cell activation and could help develop better treatments or vaccines.

Specific immune cells cause alopecia areata and blocking certain proteins can prevent it.

Expanding regulatory T cells may help treat alopecia areata by reducing harmful immune cells.

Higher free testosterone levels in men are linked to a stronger immune response, not weaker.

Certain immune cells are linked to non-scarring hair loss, suggesting potential for immune-targeted treatments.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

Dual TCR Treg cells are common in mouse tissues and vary by location.

Skin cells control immune cell placement, helping the skin respond better to challenges.

Injecting recipient splenocytes into donors' thymus can prevent graft-versus-host disease.

IL-27 may help prevent hair loss by creating immune-suppressing cells.

Anti-IL17A therapy reduced venous leg ulcer size by 64% without major side effects.

Chronic refractory alopecia areata has more skin-resident memory T cells, and JAK inhibitors may help reduce them.

γδTregs may help treat autoimmune diseases like alopecia areata by promoting hair regrowth and reducing immune attacks.

A specific group of immune and skin cells may cause chronic inflammation in atopic dermatitis.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

Special immune cells called Treg cells are important for maintaining and regenerating hair by activating a specific growth signal in hair stem cells.

Increasing Treg cells in the skin does not cure hair loss from alopecia areata in mice.

IL-19 and IL-24 help cells respond to DNA damage and could be targeted for cancer and age-related disease treatments.

Plasmacytoid dendritic cells, which overproduce IFN-α, may play a crucial role in starting alopecia areata, an autoimmune disease causing hair loss.

Hair follicles help attract immune cells to minor skin injuries.

A new mouse model effectively mimics vitiligo for research and drug testing.

Super-enhancers control CD25 expression in specific cell types, affecting immune function.

A new mutation in the STING protein causes a range of symptoms and its severity may be affected by other genetic variations; treatment with a specific inhibitor showed improvement in one patient.

Autoimmune skin diseases result from genetic and environmental factors disrupting immune checkpoints.

Higher IL-17A levels indicate more severe alopecia areata.

ILC1-like cells can independently cause alopecia areata.

Monocyte-derived dendritic cells play a key role in UVB-induced skin sensitivity and inflammation.