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Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

T cells affect skin cell genes in inflammatory diseases, and therapy can normalize these changes.

A specific type of immune cell plays a key role in causing alopecia areata and could be a target for treatment.

γδ T cells adapt uniquely to different tissues in mice.

Alopecia areata involves unique activation of certain immune cells.

IL-15 is key for T cell function and could help improve treatments for immune-related diseases.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

IL-17-producing γδ T cells help improve bone healing.

Targeting Vδ1+T-cells may help treat alopecia areata.

Lung immune cells can be trained to better fight infections after a virus.

Regulatory T cells protect hair follicle stem cells by maintaining immune privilege in the skin.

Keratin 17 is linked to various diseases, including cancer and skin conditions, and may be a target for diagnosis and treatment.

Hair follicles produce IL-7, which is essential for certain skin lymphoma cells to survive.

Regulatory T cells and mesenchymal stem cells work together to prevent immune system overreactions and tissue damage.

T cells with memory features grow in number and gather around hair follicles when there are not enough immune cells.

Enhancing Tregs can protect against alopecia areata.

Tph cells are linked to the severity of systemic lupus erythematosus.

Certain CD8+ T cells attack hair follicles in alopecia areata, suggesting they could be targeted for treatment.

CD4 T cells can cause alopecia areata by activating CD8 T cells to attack hair follicles.

Certain immune cells may cause hair loss by reacting to stressed hair follicles.

A specific immune response helps control mite populations on the skin, maintaining healthy hair follicles.

Treg dysfunction is linked to various autoimmune skin diseases, and understanding Treg properties is key for new treatments.

B cells can both help and hinder the body's defense against melanoma.

A new mutation in the TMEM173 gene and a risk allele in IFIH1 cause a unique set of immune-related symptoms.

Patients with alopecia areata have higher levels of certain immune markers, suggesting new treatment targets.

Keratin 17 is important for skin development and may help define skin cell types.

CD8+ T cells play a key role in graft-versus-host disease in certain mice models.

Skin stem cells help create protective immune cells during wound healing.

Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

Targeting TCR-Vβ2 in cutaneous T cell lymphoma shows promise for safer, more specific treatment.