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Poor response to topical immunotherapy in alopecia areata patients is linked to impaired cell responses.

A substance called poly(I:C) increases a protein called carbonic anhydrase II in skin cells, which might help with skin defense and healing.

A microneedle patch can help regrow hair by restoring immune balance in hair follicles.

Bullous pemphigoid is influenced by genetic factors, immune cell dysfunction, aging, and triggers, with treatment often improving symptoms.

A humanized CXCL12 antibody may delay and treat alopecia areata by altering the immune response.

CD8+ T cells attack hair follicle stem cells, causing scarring and hair loss.

IL-25 helps heal diabetic wounds by improving blood vessel and skin cell functions.

CD4+ skin cells may be precursors to basal cell carcinoma.

Dandruff is linked to changes in the immune system of hair follicles and skin.

Higher PD-1 levels mean fewer CD8+ T cells in alopecia areata hair follicles.

IL-9 increases skin cell movement but decreases their ability to invade, and this effect is controlled by cell contractility, not by MMPs.

Human dermal γδT-cells respond to stress in hair follicles, contributing to hair loss.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

Atopy and altered T cell functions contribute to Alopecia Areata.

Human thymus has stem cells that can self-renew and maintain their identity.

Type XVII collagen helps control skin cell growth and rejuvenates skin.

Both Th1 and Th2 immune responses are increased in alopecia areata, with Th2 response more strongly linked to how severe the disease is.

Natural killer cells play a key role in autoimmune skin diseases like pemphigus vulgaris.

Two patients with psoriasis grew extra hair after using certain psoriasis medications.

Alopecia areata involves immune system changes, especially in severe cases, with potential new treatment targets identified.

Stem cells help control the immune response to improve wound healing.

Hair follicles in nonsegmental vitiligo are better protected from immune attacks than in alopecia areata.

Activating TLR3 may help produce retinoic acid, important for tissue regeneration.

IL-27 can prevent hair loss in alopecia areata.

Hair follicles prevent NK cell attacks to avoid hair loss.

HPV16 oncogenes disrupt the normal activity of hair follicle stem cells.

Higher levels of certain immune cells in hair follicles may contribute to alopecia areata.

Alopecia areata is an autoimmune disease that targets hair follicles.

Graft-versus-host disease occurs when donor immune cells attack the recipient's body, causing skin, gut, and liver damage.

A mutation in the hairless gene speeds up severe itchy skin in mice on a special diet.