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Glucocorticoids and thyroid hormones together are essential for fetal fat development.

The HR protein's role as a repressor is essential for controlling hair growth.

Certain genetic markers can indicate higher or lower risk for systemic lupus erythematosus.

Higher IgE levels may play a role in alopecia areata, especially in males, children, and severe cases.

Transplanted hair keeps growing like it did in the donor area.

A new IL6ST gene variant may cause a unique form of hyper-IgE syndrome with skin abscesses and high IgE levels.

Discoid lupus erythematosus involves immune activation and fibrosis around hair follicles, with shared pathways across humans, dogs, and mice, suggesting potential treatments for both humans and animals.

CTHRC1 helps hair grow back, and plantar dermis mixture boosts it.

HDAC1 is crucial for skin development and preventing tumors.

Thymosin Beta 4 helps human dental pulp cells develop into tooth-forming cells, suggesting it could aid dental regeneration.

Mutations in β1 integrins cause embryonic death but have milder effects on skin.

Truncated LTBP-1 disrupts TGF-beta signaling, affecting hair growth.

High levels of HSP70 and IL-15 are linked to more severe alopecia areata.

Malt1 protease is essential for regulatory T cell function and could be targeted to boost antitumor immunity.

IL-17 is a key target for treating inflammatory skin diseases, especially psoriasis.

Your genes, especially IL-1a, can predict how well a hair growth treatment with platelet rich plasma will work for you.

Deleting Snai2 and Snai3 causes fatal autoimmunity.

Mutant stem cells adapt their metabolism differently to outcompete normal cells in the skin.

Trichohyalin is modified by enzymes to form strong structures in hair cells.

Alopecia areata patients have higher levels of certain immune receptors, suggesting new treatment possibilities.

High levels of 3 alpha-diol glucuronide in the blood are a marker of increased androgen action in women with excessive hair growth of unknown cause.

Overexpressing Dsg3 in mice skin causes excessive cell growth and abnormal skin development.

Wnt-signaling is regulated differently in skin cells and immune responses during wound healing.

TLR3 activation helps improve skin and hair follicle healing in mice.

Black hairy tongue can be an early sign of graft-versus-host disease after stem cell transplants.

The Trichodysplasia spinulosa virus protein can cause abnormal hair growth in mice.

IL-1 promotes fat cell growth in skin, while WNT inhibits it and encourages scar formation.

CD4+ skin cells may be precursors to basal cell carcinoma.