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Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

A patient's skin rash did not affect the area where a previous viral rash was healing, suggesting a rare immune response.

Controlling Tslp can improve health in AEC syndrome patients.

Targeting regulatory T cells may help treat age-related diseases.

Tongue cancer cells show more plectin-1 than non-cancer cells, but both have similar levels of trichohyalin.

IL-15 helps maintain hair growth and protects the immune status of hair follicles.

Human hair follicles have their own thyroid hormone system.

The CTLA-4 gene polymorphism does not affect polycystic ovarian syndrome.

Neurotrophin-4 increases calcium current in specific mouse neurons through the PI3K pathway.

CD34+ hair follicle stem cells can become melanin-producing cells for treating skin conditions.

Nucleic acids trigger chemokine production in skin cells, affecting skin inflammation.

P2X₃-IR fibers are widespread in rat skin and likely help detect pain.

Melanocytes may trigger the immune response in alopecia areata, affecting hair regrowth.

PCOS involves immune and genetic factors, with key roles for T cells and specific genes.

Increasing regulatory T cells may help treat alopecia areata by reducing autoimmunity and promoting hair growth.

The medicine Cyclosporin A might cause excessive hair growth by reducing a protein that controls hair growth.

Cyclosporin A, a drug, reduces TGF-β2 expression in skin cells, potentially causing excessive hair growth through a process involving the calcineurin/NFAT pathway.

Alopecia areata is linked to reduced T cell function and auto-immunity.

TNFα helps grow and maintain liver cells in 3D culture for a long time.

Blocking interferon-gamma helps prevent and treat hair loss in Alopecia Areata.

The HCR gene contributes to psoriasis risk.

Patients with certain FoxN1 gene mutations have severe immune issues but normal skin and hair.

IL-4 gene variation may increase the risk of alopecia areata in Turkish people.

Skin-resident memory T cells may contribute to chronic alopecia areata and baricitinib could be a potential treatment.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

Targeting METTL1 may help slow papillary thyroid cancer growth and spread.

CD2 might be a new treatment target for patchy alopecia areata.