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CD8+ T cells expand significantly in alopecia areata, suggesting new treatment targets.

Increasing Smurf2 hinders hair follicle stem cell differentiation and wound healing.

The skin can independently form immune responses through special structures, offering new ways to treat skin diseases.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

sPLA2-IIA increases growth in hair follicle stem cells and cancer cells, suggesting it could be targeted for hair growth and cancer treatment.

TGF-β2 helps yak hair follicles enter the regression phase, while HSP70 tries to prevent it.

Finasteride increases CD8+ T cells in BPH tissues.

Skin cells and immune cells change in a skin condition called hidradenitis suppurativa, and a certain treatment can improve these changes.

TH antibodies in vitiligo and AA patients recognize the same protein parts.

A mouse model for studying scleroderma in chronic graft-versus-host disease was successfully created.

Plasmacytoid dendritic cells, which overproduce IFN-α, may play a crucial role in starting alopecia areata, an autoimmune disease causing hair loss.

Innate lymphoid cells type 1 may contribute to alopecia areata.

c-Maf positive macrophages help form hypertrophic scars by affecting fibroblasts and collagen production.

Type 2 diabetes slows down skin and hair renewal by blocking important stem cell activation in mice.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.

OCT4B1 and SOX-2 levels are higher in Crohn's and ulcerative colitis patients, possibly aiding tissue repair.

iNKT cells help develop and maintain healthy skin in young mice.

Regulatory T cells help hair growth by using the Cxcr4-Cxcl12 pathway.

A new gene mutation is linked to monilethrix in the studied family.

Ptch2 plays a key role in controlling stem cell function and the ability to regenerate after birth.

Targeting regulatory T cells may help treat age-related diseases.

NKG2D+CD4+ T cells are higher in alopecia areata patients and may be involved in the disease.

IL-17 is more important than IFN-γ in causing severe hair loss in chronic alopecia areata.

The study suggests that a specific type of immune cell, memory-like NK cells, may increase during active hair loss in Alopecia areata.

Thymosin beta-4 promotes hair growth in mice.

The E2 protein affects gene activity in hair follicles of mice.

The new antibody, TYHF-1, specifically targets certain hair-related structures.

These ovarian conditions cause high testosterone levels.