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Different species and human skin models vary in their skin enzyme activities, with pig skin and some models closely matching human skin, useful for safety assessments and understanding the skin's protective roles.

Scientists can control how skin stem cells divide by using different treatments.

Stem cell activity influences autoimmune disease outcomes by affecting immune responses and tissue regeneration.

The research found that a protein called PPARg is important for the formation and healing of sebaceous glands, which can regenerate independently from hair follicles.

5α-reductase 2 is crucial for stress response in male rats.

Low humidity harms skin health by damaging the skin barrier and increasing inflammation.

Proper mTOR signaling is crucial for healthy skin and preventing skin diseases.

Damaged skin has a weakened barrier, making it more vulnerable to substances and inflammation.

People with Hidradenitis Suppurativa have less diverse skin bacteria and different bacterial metabolism than healthy individuals.

Sensory neuron and Merkel cell changes in the skin happen independently during normal skin maintenance.

Lower TGFß1 levels help stem cells become beige fat cells.

The Glu413Lys mutation in keratin affects hair stability, while Glu413Asp does not.

Reactive oxygen species are key for parasitic plant attachment and affect root development in various plants.

Targeting specific metabolic and ionic pathways may improve alopecia areata treatment.

Low selenium levels can extend lifespan but worsen health issues.

High selenium exposure can harm thyroid function, immune system, liver, skin, and may increase neurotoxicity and cancer risk.

Selenium is essential but can be harmful in excess, and China struggles with balancing its levels for health.

Finasteride irreversibly affects human steroid 5α-reductase 2, providing insight into its catalytic mechanism and disease-related mutations.

Chronic stress can reduce skin pigmentation.

Aging reduces skin cell renewal and defense against germs due to TGFbeta, but blocking TGFbeta could help restore these functions.

Weak cell junctions disrupt hair follicle stem cell rest.

The microenvironment controls adult stem cells' behavior and fate.

Melanocyte stem cells in hair follicles are key to understanding and potentially preventing hair graying.

Red light exposure can slow aging in mice by improving fat metabolism.

A new light-activated treatment speeds up healing of infected wounds without antibiotics.

Oxidative stress causes hair graying by damaging hair follicle melanocytes more than skin melanocytes.

Dihydrotestosterone can be made from dehydroepiandrosterone in skin cells without needing testosterone.

Acid inside cells speeds up aging and turns on aging signs in mice.