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Cell-based therapy using specific immune cells may help treat alopecia areata by promoting hair regrowth.

Tsc2-deficient stem cells can help understand and treat TSC-related tumors.

Human hair follicle stem cells can become smooth muscle cells using specific growth factors.

Mouse skin cancer progression involves a unique group of cells marked by ABCG2 and MTS24.

Increasing Treg cells in the skin does not cure hair loss from alopecia areata in mice.

Human sweat glands have a type of stem cell that can grow well and turn into different cell types.

Recombinant thymosin β4 promotes blood vessel growth and reduces muscle damage.

Cells from skin and lung can help regenerate hair follicles.

Skin cells control immune cell placement, helping the skin respond better to challenges.

T cells and inflammation are crucial in atherosclerosis, with anti-inflammatory treatments showing promise.

Hair follicle stem cells use specific chromatin changes to control their growth and differentiation.

Scientists used a special imaging technique to observe that hair follicle regeneration involves cell division and structural changes, mostly in the lower part of the follicle, and that the dermal papilla at the base is crucial for regrowth.

β-thymosin in invertebrates is more complex and diverse than in vertebrates.

Different types of inactive melanocyte stem cells exist with unique characteristics and potential to develop into other cells.

Sox9 is crucial for hair follicle stem cells to become melanocytes instead of glial cells.

Hair follicle stem cells can become heart muscle cells.

Boosting certain cell signals can prevent hair loss from chemotherapy and radiation.

PCFCL may have unrecognized subtypes and needs more research.

Certain immune-related proteins are higher in people with alopecia and their healthy relatives, hinting at a genetic link.

The Hippo signaling pathway helps control organ size during regeneration by regulating gene expression.

IL-1 and IL-7 help activate cells that boost hair follicle stem cell growth, aiding wound healing.

The role of γδT-cells in causing alopecia areata remains unclear.

Folliculotropic mycosis fungoides has unique molecular features and cell interactions that could guide targeted therapy.

Certain CD8+ T cells attack hair follicles in alopecia areata, suggesting they could be targeted for treatment.

Thyroid hormones affect hair follicle stem cells by promoting differentiation and reducing growth.

Myeloid cells can turn into skin and hair cells to help heal wounds.

Bone marrow-derived cells contribute to skin tumors, suggesting new treatment targets for non-melanoma skin cancers.

Lipid-antigen stimulation may play a role in folliculotropic mycosis fungoides.

Overexpressing Tβ4 in cashmere goats improves hair fiber traits and increases cashmere yield.

BLIMP1 is essential for skin maintenance but not for defining sebaceous gland progenitors.