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Alpha-difluoromethylornithine prevents cancer in mice but causes hair loss.

A new gene, mrp4, is found in mice and may play a unique role in hair follicle development in tails and ears.

Cathepsin L deficiency causes hair and skin issues in mice.

Sox13 is a marker for early hair follicle development but not essential for skin and hair growth.

The study created a mouse model that survives longer and shows fewer symptoms of pemphigus vulgaris.

TCDD changes skin cell growth and keratin production in mice.

The study created a mouse model to mimic degenerative diseases for testing tissue repair and new therapies.

Scientists made a mouse that can be made to lose hair and then grow it back.

Lack of KSR1 stops certain skin tumors in mice.

Transferrin receptor 1 is essential for normal function in the intestines, pancreas, and skin.

Reducing Tyrosinase prevents mature color pigment cells from forming in mouse hair.

Some thymic peptides can increase human hair growth, while others may inhibit it.

The PP2A-B55α protein is essential for brain and skin development in embryos.

Removing the p75 gene in mouse skin cells didn't affect their skin or hair growth.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

The STIM1 R304W mutation in mice leads to bone changes and teeth hair growth.

PTHrP and its receptor can control blood vessel growth and hair development in mouse skin.

The new Hairless R/J mice model improves imaging for tumor monitoring and cancer therapy evaluation.

Overexpression of PKCepsilon leads to increased TNFalpha, promoting metastatic squamous cell carcinoma in mice.

Tabby mutations in mice affect hair follicle development and help study genetic mapping and certain medical conditions.

Thy-1 protein helps improve blood flow and wound healing in the skin.

HtrA2 activity is crucial for normal hair growth by regulating fat cell development.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

Overexpressing the human H-ferritin gene in mice causes mild growth delay and temporary hair loss.

Introducing the OTC gene improved symptoms in mice with OTC deficiency.

Removing both Atr and Trp53 genes in adult mice causes severe tissue damage and death due to DNA damage.

The mouse model showed defects in adult stem cell maintenance related to Hutchinson-Gilford progeria syndrome.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

A gene mutation in mice causes increased mast cells and disorganized hair follicles in their skin.