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A new mutation in the STING protein causes a disease with lupus-like symptoms and responds well to a specific inhibitor treatment.

Hair transplantation successfully treated hair loss in a patient with Trichorhinophalangeal syndrome.

Alternative treatments are needed when Tofacitinib alone fails for alopecia areata.

FTY720 helps transplanted fat survive better by reducing immune rejection and improving blood vessel growth.

Tofacitinib can cause a rare rash called SDRIFE, which resolves after stopping the drug.

Combining PRP with Tressfix Serum is more effective for hair regrowth in telogen effluvium than using either alone.

Tofacitinib helped a patient regrow hair after 40 years of total hair loss.

Continued ritlecitinib treatment helps sustain hair regrowth in alopecia areata patients.

Tiger tail banding and hair abnormalities are reliable indicators for diagnosing trichothiodystrophy.

The assay effectively identifies compounds that affect immune cell activation.

Autologous micrografting via the Rigenera® system shows promise in improving hair loss treatment.

Contact immunotherapy can change immune responses in alopecia areata, suggesting new treatment targets.

Gene therapy in mice increased lifespan and improved health without causing cancer.

The early genes of a specific virus can cause abnormal skin cell growth and hair follicle changes.

Continued ritlecitinib treatment can improve hair regrowth in some alopecia areata patients who initially don't respond.

Beta-trace protein may help diagnose and predict treatment response in certain nerve disorders.

Cognitive-behavioral therapy with habit reversal training is the most effective treatment for trichotillomania.

Human dermal γδT-cells respond to stress in hair follicles, contributing to hair loss.

Oral tacrolimus helped a rheumatoid arthritis patient regrow hair lost due to alopecia areata.

A child with trichothiodystrophy also had autoimmune thyroiditis and anemia, which is a new finding.

Low TRPS1 expression in skin and hair cells is linked to hair problems in Trichorhinophalangeal syndrome.

TNFα and interleukin-1 blockers reduce skin inflammation from EGFR antibody therapy.

Adding TERT and BMI1 to certain skin cells can improve their ability to create hair follicles in mice.

Thymic mesenchymal cells have unique gene expression that supports their specific functions in the thymus.

Injecting recipient splenocytes into donors' thymus can prevent graft-versus-host disease.

The NUDT15 R139C variant causes thiopurine-induced leukocytopenia through a different mechanism than previously thought in Japanese patients with inflammatory bowel disease.