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Increased TEMRA cells can predict treatment outcomes in rapidly progressive alopecia areata.

Immune cells in Hidradenitis suppurativa become more inflammatory and may be important for treatment targets.

Tgm2 helps stabilize dying cells and aids fibroblast attachment to the extracellular matrix.

ADM scaffolds help skin heal by promoting a healing-type immune response.

NDP-MSH protects brain cells and reduces inflammation in Parkinson's disease by activating MC1R and involving Tregs.

BST2 is highly expressed in certain immune cells in alopecia areata, suggesting a role in the disease.

Immune cells around hair follicles help control hair growth and could be targets for treating hair disorders.

Regulatory T cells and mesenchymal stem cells work together to prevent immune system overreactions and tissue damage.

Single-cell sequencing shows that different types of macrophages have unique roles in wound healing.

A parasite-derived molecule speeds up skin healing and affects immune cell behavior without increasing scarring.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

Restoring nerve-macrophage communication may help treat autoimmune diseases.

The gene Tfap2b is essential for creating a type of stem cell in zebrafish that can become different pigment cells.

Trichoblastomas may mimic fetal skin development by having many Merkel cells, unlike adult skin.

Targeting adipocyte-to-mesenchymal transition could help treat fibrosis.

TSC2-/meth cells can cause skin lesions, hair growth, and lung issues, and may be treated with chromatin remodeling agents.

IL-17 plays a key role in severe hair loss in chronic alopecia areata.

HAT-MSCs can effectively engulf harmful microbes and particles, aiding infection treatment.

HtrA2 activity is crucial for normal hair growth by regulating fat cell development.

Hair follicle development and microbes help regulatory T cells gather in newborn skin.

Inhibiting IL-17 and IL-23 improves wound healing in obese, diabetic mice by promoting healing macrophages.

In alopecia areata, certain immune cells increase and express a protein linked to immune activation.

Microglia are essential for zebrafish brain repair by controlling inflammation.

Engineered vesicles from macrophages help hair growth in mice and humans.

CD206+ macrophages are crucial for hair growth in alopecia areata treatment.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

Blocking androgen receptors boosts macrophages' ability to clear certain bacteria.

A parasite molecule can speed up skin healing and reduce scarring.

Lung immune cells can be trained to better fight infections after a virus.