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Deleting the p63 gene in certain cells causes problems in thymus development and severe hair loss in mice.

Reduced TRPS1 leads to increased STAT3 and SOX9 in hair follicles, affecting hair growth.

TRPS1 is crucial for bone, kidney, and hair follicle development.

ΔNp63α helps control a protein that stops cancer cells from spreading.

p63 controls Satb1 to help skin develop properly.

Trps1 is essential for proper hair follicle development.

P63 is a marker for epidermal stem cells in rats.

p63 and p73 regulate the vitamin D receptor, affecting cancer cell behavior and vitamin D sensitivity.

mrp/plf-mRNA can indicate tumor-promoting effects in skin.

TR3 is mainly found in hair follicle stem cells and may be involved in hair loss.

Trps1 plays a key role in hair follicle development and cycling.

p63 may influence skin cancer development and cell differentiation.

A small change in the TRPS1 gene leads to a less severe form of a syndrome affecting hair, nose, and finger development.

Low-level laser therapy helps hair growth and reduces hair loss with few side effects.

Mutations in the p63 gene affect skin adhesion, barrier integrity, and hair growth.

The Itpr3 gene causes a specific hair pattern in mice.

TRF1 is crucial for creating and maintaining stem cells and marks both pluripotent and adult stem cells.

A new mutation in the TRPS1 gene was found in a Ukrainian girl with Trichorhinophalangeal syndrome type I.

TRPS1 loss in balding scalp areas contributes to hair loss in androgenic alopecia.

A position effect on the TRPS1 gene causes excessive hair growth in humans and mice.

The protein p53 directly reduces the production of Keratin 17, a skin and hair protein, in rats with radiation dermatitis.

Low TRPS1 expression in skin and hair cells is linked to hair problems in Trichorhinophalangeal syndrome.

A new mutation in the TRPS1 gene caused Trichorhinophalangeal syndrome in a 17-year-old, highlighting the need for genetic testing.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

Flutamide combined with an LHRH agonist effectively inhibits prostate growth, suggesting it could treat prostate cancer.

Strong skin creams work well for long-term scalp inflammation in Rapp-Hodgkin Ectodermal Dysplasia.

Testosterone affects androgen receptors and lipid storage in cells, while DHEA does not convert to testosterone or affect these receptors in the same way.

The created skin model with melanoblasts improves the study of skin color and offers an alternative to animal testing.

A woman with a unique syndrome similar to TRPS has a genetic change near the TRPS1 gene, affecting its regulation.