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Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

Thy-1 protein helps improve blood flow and wound healing in the skin.

Targeting THY1 can improve skin repair and healing.

VEGFR-2 is active in hair follicles, sebaceous glands, sweat glands, and skin on the human scalp.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

A mycobacterial protein shares a similar region with a human skin protein, possibly affecting skin diseases.

Acute stress increases Y1 receptor gene expression in certain brain areas, but repeated stress does not.

Human skin cells can respond to thyroid-stimulating hormone, affecting hair and skin health.

Dermal papilla cells mainly drive blood vessel growth in hair follicles.

Expressing the human vitamin D receptor in skin cells prevents hair loss in certain mice.

γδ T cells are crucial for early wound healing after a skin virus infection.

Invasomes with natural terpenes can improve drug delivery through the skin.

Controlling transposable elements is crucial for successful tissue regeneration.

A new gene, mrp4, is found in mice and may play a unique role in hair follicle development in tails and ears.

Keratin 79 cells help form and regenerate hair canals.

High testosterone affects blood vessel function, with estrogen playing a key role in maintaining heart health.

LRIG1 protein affects hair growth by regulating skin receptors, leading to hair loss when overexpressed.

Prostaglandin E2 affects human platelet activity in complex ways that could lead to personalized heart disease treatments.

Tongue cancer cells show more plectin-1 than non-cancer cells, but both have similar levels of trichohyalin.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

The virus linked to a rare disease was found in a patient's blood and urine before skin symptoms appeared.

Serotonin helps hair grow by activating certain cells.

Tmem50b and 2610305D13Rik genes play key roles in early mouse embryo development.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Zebrafish with mutations similar to human Cantú Syndrome have heart cells with altered channel properties, making them a good model to study the condition.

Nitrate helps plants manage phosphate uptake and starvation responses through NIGT1 proteins.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.