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Herbal compounds like ricinoleic acid, quercetin-3-O-rutinoside, and hinokiflavone may be safe and effective for treating hair loss.

Researchers created five new human scalp cell lines that could be useful for hair growth and loss research.

Exosomes from fat-derived stem cells help repair large bone defects by attracting and enhancing bone marrow stem cells.

High-concentration cell-free adipose extract reduces scar formation and improves scar appearance.

Chewing betel nut may increase the risk of type 2 diabetes, and saw palmetto can improve urinary symptoms in BPH with fewer side effects but may cause bleeding and allergic reactions.

Diphencyprone initially increases mouse hair growth, then slows it, possibly due to changes in specific protein levels.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

Overexpressing GLI-1 in mice skin can cause tumors like human basal cell carcinomas.

Boosting certain cell signals can prevent hair loss from cancer treatments.

Atg5 can promote tumors when autophagy is deficient but suppresses them under normal conditions.

Some viruses can cause cancer by changing cell processes and avoiding the immune system; vaccines and targeted treatments help reduce these cancers.

Deleting TNFα gene reduces skin cancer risk in certain mice.

Childhood and adult height, and early immune-related hospitalizations increase testicular cancer risk, while baldness, larger families, and sports reduce it.

Noggin promotes skin tumors by activating Wnt and Shh pathways.

Researchers developed promising agents for prostate cancer imaging, with the best one showing high potential for clinical use.

Cancer risk is linked to the balance of mutations and environmental factors, not just the number of mutations.

Keratin 10 end domains may increase skin cancer risk by reducing cell death.

Etoposide makes radiation therapy more effective against tumors without major side effects.

Overexpression of E2F1 can lead to skin tumors and disrupt hair growth.

Long-standing benign tumors can become cancerous, especially in people with weakened immune systems.

Activating c-Myc in skin causes rapid cell growth and changes, but these effects are reversible.

TS-13 effectively stops tumor growth and reduces chemotherapy side effects.

Higher TGF-β signaling may increase skin cancer risk in organ transplant recipients.

Lack of vitamin D receptor increases skin tumor risk by boosting hedgehog signaling.

Tumor proteins can both promote and suppress cancer, depending on the situation.

HOTTIP and miR-10b contribute to glioma therapy resistance by affecting cell behavior, suggesting they could be targets for treatment.

Mitochondria may influence how cells respond to radiation, affecting nearby non-irradiated cells.

The type of tumor suppressor gene lost affects the behavior of skin cancer.

Disrupting Stat3 in hair follicle stem cells greatly reduces skin tumor formation.

BMP signaling helps prevent skin tumors by blocking cancer-promoting pathways.