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Phosphorylation controls TFEB's location in the cell, affecting cell metabolism and stress response.

The L412F variant of TLR3 is linked to skin infections, more viral infections, and autoimmune issues.

Non-coding RNA boosts retinoic acid production and signaling, aiding regeneration.

A specific mutation in PA-PLA1α causes abnormal hair growth.

Folliculin helps regulate energy and nutrient sensing, impacting Birt–Hogg–Dubé syndrome.

Overexpression of hurpin in mice leads to abnormal skin and higher skin cancer risk.

Genotyping for NUDT15 p.Arg139Cys can help predict thiopurine side effects in Japanese IBD patients.

QSOX enzymes help form protein bonds in cells, especially in tissues with high secretory activity.

P2X₃-IR fibers are widespread in rat skin and likely help detect pain.

The SLICK1 allele in Holstein heifers affects hair and immune traits without altering prolactin signaling.

Hsc70 protein may influence hair growth by responding to androgens.

FP-1 is a key protein in rat hair growth, active only during the growth phase.

Higher activity in lichen planopilaris is linked to certain immune and tissue genes.

The B2 genes are crucial for hair growth in rats.

STAT3 reduces FST gene activity and cell growth, affecting hair development and wool quality.

The report expanded knowledge of MBTPS1-related disorders by identifying new symptoms.

ULBP3 could be a marker for diagnosing alopecia areata incognita and may be linked to its cause and development.

Inhibiting ALOX12 can help hair cuticle maturation by increasing S100A3 citrullination.

LPA3 is crucial for embryo implantation and links LPA to prostaglandin signaling.

ATP-dependent chromatin-remodeling complexes are crucial for gene regulation, cell differentiation, and organ development in mammals.

SOCS1 and SOCS3 help control skin inflammation and are important for developing treatments for skin diseases.

MPZL3 protein helps keep sebaceous gland size and cell growth in check.

The protein called small nuclear ribonucleoprotein polypeptide E is identified as a cause of a type of hair loss without other symptoms.

A gene mutation in mice causes severe skin disorder similar to a human condition.

Toll-like receptor 3 helps repair the skin barrier after UV damage.

Increased 14-3-3 proteins may block hair cycle regression, causing hair loss.

CTIP2 may help in skin development and maintenance.

PRC2 is essential for maintaining intestinal cell balance and aiding regeneration after damage.

Par3–mInsc and Gαi3 work together to ensure proper cell division orientation in skin development.