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The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

Understanding cellular interactions in VCA may lead to better treatments and reduce rejection.

Ritlecitinib reduces alopecia areata symptoms by blocking JAK3/TEC signaling and T-cell activity.

Skin's Regulatory T cells are crucial for maintaining skin health and could be targeted to treat immune-related skin diseases and cancer.

A new test helps find drugs to treat head and neck cancer by targeting c-Rel.

Dysplastic nevi have unique gene expressions, making them distinct from common melanocytic nevi.

Super-enhancers control CD25 expression in specific cell types, affecting immune function.

Cells that move well may improve hair loss treatments by entering hair follicles.

Tofacitinib and low-dose IL-2 may help maintain hair regrowth in alopecia areata without ongoing treatment.

Highly active but fewer CD14+CD16- monocytes are found in Alopecia Areata patients, regardless of severity.

CD133+ progenitor cells have therapeutic potential for diabetic ulcers and heart attack recovery, with manageable risks.

People with myotonic dystrophy type 1 have a higher chance of getting skin tumors, including melanoma.

S-1 treatment led to a complete response in pancreatic cancer with manageable side effects.

A child with trichothiodystrophy also had autoimmune thyroiditis and anemia, which is a new finding.

IL-13 protein is much higher in the skin of atopic dermatitis patients than in healthy skin.

AHST shows promise for treating type 1 diabetes but needs more research before widespread use.

Tildrakizumab showed limited effectiveness in treating chronic alopecia areata in a small group of patients.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

A new mutation in the STING protein causes a disease with lupus-like symptoms and responds well to a specific inhibitor treatment.

IL-17C is important in inflammatory skin diseases and could be a target for treatment.

ILC1 cells contribute to hair loss in alopecia areata.

Activating TLR9 helps heal large wounds and regrow hair by involving a specific type of immune cell.

Skin cells and immune cells change in a skin condition called hidradenitis suppurativa, and a certain treatment can improve these changes.

Alopecia areata is linked to reduced T cell function and auto-immunity.

Hair follicles help skin immune recovery after UVB exposure.

Innate lymphoid cells type 1 may contribute to alopecia areata by damaging hair follicles.

Monocyte-derived dendritic cells play a key role in UVB-induced skin sensitivity and inflammation.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

A specific gene mutation causes vitamin D resistance, but certain vitamin D analogs might help.