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A rare case of a transplant patient developing a skin condition linked to HPV-49.

Dihydroxyacetone in self-tanning lotion helped reduce sun-related skin issues in a woman with variegate porphyria.

The study suggests a link between varicella-zoster virus and segmental vitiligo, with evidence of the virus disrupting skin pigment cells.

VDR gene variations may affect carbohydrate metabolism in young women with hyperandrogenism.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

A vitamin D receptor mutation causes rickets and affects immune responses.

Early diagnosis and teamwork are crucial for managing ILVASC effectively.

The DLQI is a key tool for measuring quality of life in dermatology.

Vitamin D may help treat hair disorders.

Understanding the 2D:4D digit ratio in vitiligo patients may help in clinical assessments.

Vitamin D receptor helps prevent skin tumors.

The method accurately measures vinflunine in dog plasma.

CaBP1 and CaBP2 are necessary for proper hearing and neurotransmission in the ear's inner hair cells.

CaBP1 and 2 are important for maintaining the activity of calcium channels necessary for hearing in inner ear cells.

CaBP1 and CaBP2 are necessary for proper hearing and neurotransmission in the ear's inner hair cells.

Vinblastine can help treat childhood ITP but may cause reversible side effects.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

VDAC2 promotes cell death in cashmere goat hair follicles through the P53 pathway.

Laser Doppler velocimetry is a valuable noninvasive tool for skin blood flow research in dermatology.

Vitamin D receptor helps protect skin cells from UV damage and supports their growth.

The document concludes that various skin conditions have specific characteristics and treatments, and highlights the importance of vitamin D in managing these dermatological issues.

Early detection of specific VDR mutations is crucial for effective treatment and better dental outcomes in children with hereditary vitamin D–resistant rickets.

Vitamin D receptor may help protect against UV-induced skin cancer.

Vitamin D receptor is essential for preventing hair loss by regulating hair cycle stages.

Vitamin D Receptor is crucial for hair follicle shrinkage and cell death, affecting hair growth.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

Ex vivo dermatoscopy may lower lab costs and improve diagnosis speed for hair loss biopsies.