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The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

A new gene mutation causes vitamin D resistance and hair loss in two unrelated girls.

Vitamin D receptor is crucial for healthy hair growth and preventing hair loss.

Alopecia in VDR knockout mice is due to impaired hair cycle initiation, not keratinocyte issues.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.

Targeting the Wnt pathway might help treat hair loss.

Expressing the human vitamin D receptor in skin cells prevents hair loss in certain mice.

Low vitamin D receptor levels found in hair loss patients; topical vitamin D treatment suggested.

Vitamin D boosts a specific protein in skin cells linked to hair follicles.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Vitamin D receptor gene variations are not linked to alopecia areata.

A vitamin D receptor mutation causes rickets and affects immune responses.

Combining skin tissue pathology with genetics has greatly improved the diagnosis and understanding of certain skin diseases.

Early detection of specific VDR mutations is crucial for effective treatment and better dental outcomes in children with hereditary vitamin D–resistant rickets.

Three new gene mutations cause rickets and hair loss, treatable with high calcium and calcidol, but hair regrowth is rare.

Vitamin D receptor FokI gene variation is not linked to alopecia areata.

Vitamin D receptor helps protect skin cells from UV damage and supports their growth.

Vitamin D receptor helps prevent skin tumors.

Retinoic acid affects skin and hair health by working with specific receptors, and its absence can lead to hair loss and skin changes.

Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Vitamin D receptor and mediator 1 are crucial for healthy skin and hair growth.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

A mutation in the hairless gene speeds up severe itchy skin in mice on a special diet.

Vitamin D helps regulate calcium levels in the body.

Vitamin D helps skin stem cells heal wounds by working with a key skin protein.

Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.