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A mutation in the vitamin D receptor causes severe resistance to vitamin D, affecting bone health but not hair growth.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Vitamin D and its receptor are essential for hair growth, cell regulation, immune function, and heart health.

The vitamin D receptor can regulate genes and affect hair and hormone levels without its usual activator.

A girl had rickets due to a gene mutation affecting vitamin D response.

A new gene mutation causes vitamin D resistance and hair loss in two unrelated girls.

The review found no clear link between vitamin D receptor gene variations and polycystic ovary syndrome.

Hairless protein is crucial for healthy skin and hair, and its malfunction can cause hair loss.

People with alopecia areata often have lower vitamin D levels, which are linked to more severe and longer-lasting hair loss, but vitamin D receptor levels in the skin don't show the same pattern and don't predict treatment success.

Vitamin D receptor is crucial for healthy hair growth and preventing hair loss.

Alopecia in VDR knockout mice is due to impaired hair cycle initiation, not keratinocyte issues.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Vitamin D is crucial for bone health and preventing serious diseases.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

The vitamin D receptor can affect gene activity even without its usual hormone in hair and skin.

Targeting the Wnt pathway might help treat hair loss.

Expressing the human vitamin D receptor in skin cells prevents hair loss in certain mice.

A single amino acid change in the vitamin D receptor can disrupt its function and lead to hair loss.

Low vitamin D receptor levels found in hair loss patients; topical vitamin D treatment suggested.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

Different Hairless isoforms affect Vitamin D receptor activity in hair regulation, with one repressing and the other stimulating it.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.