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Vitamin D receptor is important for regulating hair growth and wound healing in mice.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

Targeting the Wnt pathway might help treat hair loss.

Expressing the human vitamin D receptor in skin cells prevents hair loss in certain mice.

Different Hairless isoforms affect Vitamin D receptor activity in hair regulation, with one repressing and the other stimulating it.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

Vitamin D and calcium are important for skin stem cell function and wound healing.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Vitamin D receptor gene variations are not linked to alopecia areata.

A vitamin D receptor mutation causes rickets and affects immune responses.

Combining skin tissue pathology with genetics has greatly improved the diagnosis and understanding of certain skin diseases.

Early detection of specific VDR mutations is crucial for effective treatment and better dental outcomes in children with hereditary vitamin D–resistant rickets.

Three new gene mutations cause rickets and hair loss, treatable with high calcium and calcidol, but hair regrowth is rare.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Vitamin D receptor FokI gene variation is not linked to alopecia areata.

Vitamin D receptor helps protect skin cells from UV damage and supports their growth.

Vitamin D receptor helps prevent skin tumors.

Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

Certain gene variations in the Vitamin D receptor may increase the risk of chronic hair loss.

Hairless protein helps control hair growth by regulating vitamin D receptor activity.

Vitamin D helps regulate calcium levels in the body.

Patients with the same vitamin D receptor mutation showed different symptoms due to other factors.

Vitamin D and its receptor are important for hair and scalp health and may help treat hair loss.

Removing the VDR gene in skin cells reduces their growth and affects hair-related genes.

Vitamin D and its receptor help regulate skin functions like cell growth, immunity, and hair cycle.

Vitamin D helps skin stem cells heal wounds by working with a key skin protein.

Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.

Key genes and pathways influence cashmere production in goats.