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A rare genetic disease causes rickets and often hair loss in young children, which can improve with specific treatments.

Two siblings with a rare type of rickets and hair loss had low vitamin D levels, which is not typical for their condition.

Growth hormone therapy can improve symptoms and growth in children with Vitamin-D Dependent Rickets Type-2.

Calcium supplements improved bone deformities but not skin papules or hair loss.

Vitamin D Dependent Rickets Type-2A can cause complete hair loss and bone growth issues in infants.

Measuring 24-OHase induction helps identify defects in vitamin D processing and predict treatment response.

DDR-II hair follicle cells don't respond to 1, 25-dihydroxyvitamin D3, aiding diagnosis.

A child with a rare vitamin D-resistant condition improved with treatment.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

A child with rickets and hair loss might have a rare type of rickets that doesn't improve much with usual vitamin D and calcium treatment.

A 3-year-old with vitamin D-resistant rickets and severe hair loss died despite treatment, highlighting the need for improved management of the condition.

A 2-year-old girl with a rare vitamin D disorder had rickets and hair loss, but treatment was ineffective due to poor compliance.

Vitamin D-dependent rickets Type II causes bone problems and hair loss, and doesn't improve with Vitamin D treatment.

VDDR I and II are genetic disorders affecting vitamin D use, causing rickets, with VDDR I treatable by vitamin D supplements and VDDR II needing high doses and calcium.

Nocturnal calcium infusions improved a boy's severe rickets without causing hair loss.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

Hair loss can be a key sign of a rare type of rickets when vitamin D treatment doesn't work and advanced tests aren't available.

Patients with this syndrome can have different responses and worsening resistance to treatment over time.

Intravenous and oral calcium effectively treated vitamin D dependent rickets type II, improving growth and bone health.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requires high doses of calcium and vitamin D for management.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

Child with rickets improved with a specific vitamin D therapy, but alopecia did not change.

Vitamin D is crucial for bone health and preventing serious diseases.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

Patients with the same vitamin D receptor mutation showed different symptoms due to other factors.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

The research showed that Vitamin D and its receptor are important for healthy bones and normal hair and skin in rats.

A 4-year-old boy with a rare type of rickets and hair loss improved in bone health but not hair growth after vitamin D and calcium treatment.

A boy's hair loss and bone issues improved with specific vitamin D treatment and calcium.