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Treatments with sunscreen and active ingredients can reduce skin hyperpigmentation by decreasing DNA methylation.

Vitiligo is caused by the immune system attacking skin pigment cells.

New vitiligo treatments focus on controlling immune damage and restoring skin color.

Betamethasone activates and increases the growth of certain skin cells from hair follicles.

Vitamin C deficiency changes gene expression, affecting skin and hair health.

People with chronic hair loss may have a higher chance of Vitamin B12 deficiency.

Iron supplements combined with Quilib lotion effectively treat hair loss in people with iron deficiency anemia.

VEGF helps hair grow and determines follicle size by increasing blood vessel growth.

Mast cells and the CB1 receptor may be key in causing IgA vasculitis.

Compound Baqia Danshen Liniment may effectively treat vitiligo by improving skin and hair pigmentation and reducing inflammation.

Dexamethasone reduces inflammation and delays skin cell changes caused by TPA.

Aminoguanidine increases a specific growth signal in stored hair grafts, which may help them survive better after being transplanted.

Zinc deficiency might affect vitamin B12 levels.

Fat tissue extract may help treat vitiligo by reducing cell stress and promoting skin repair.

Indinavir can cause hair loss and other skin problems.

The enzyme RDH12 plays a role in vision and retinal disease, with mutations leading to early onset visual loss and blindness, but the exact disease mechanism is unclear and there are no treatments yet.

Vitamin C derivative increases versican in cells, potentially aiding hair growth.

Segmental Vitiligo is a stable, early-onset form of vitiligo that responds well to early treatment and is ideal for repigmentation studies.

Ivarmacitinib effectively regrows hair in severe alopecia areata.

Severe vitamin deficiencies in children can cause significant hair problems.

New treatments for vitiligo, including the first FDA-approved option, are improving patient care and quality of life.

Targeting TCR-Vβ2 in cutaneous T cell lymphoma shows promise for safer, more specific treatment.

The hydrogel helps wounds heal without scars and promotes new hair growth.

Targeting specific T cells may help treat alopecia areata.

Excess vitamin A can cause hair follicles to change into gland-like structures.

The TRPV3 channel structure changes linked to severe itch and hyperkeratosis were identified using cryo-EM.

Vitamin A levels don't significantly affect alopecia areata.

Vitamin C derivative reduces hair loss-related protein in cells.

More effective treatments are needed for alopecia areata, hidradenitis suppurativa, and vitiligo.