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Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

Mutations in the LIPH gene cause woolly hair in a child.

Different diets change the fat composition in mouse skin, often reducing beneficial omega-3 fatty acids.

Mutations in specific genes cause certain congenital defects in dogs, aiding in understanding similar human diseases.

Lgr5 and the vitamin D receptor are key in controlling skin inflammation and tumor risk in mice.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

Gorillas in Gabon improved after protein supplements were added to their diet.

Tea polyphenol–zinc helps protect the liver and intestines from oxidative stress.

Mouse stem cells from hair follicles can improve wound healing and reduce scarring.

Old treatments for other skin conditions showed promise for hair regrowth in mice with a hair loss condition.

A new mouse mutation causes skin and hair issues, influenced by another gene.

Regulatory T cells are essential for normal and improved wound healing in mice.

Removing certain immune cells in mice causes their hair to enter the growth phase earlier than usual.

Mouse skin and hair aging starts at 200 days, with changes in hair follicles and more white hairs as signs of aging.

Rapamycin treatment helps with mitochondrial disease by reducing PKC levels.

A vitamin D receptor mutation causes rickets and affects immune responses.

Tooth loss can cause premature gray hair due to less chewing.

Blocking DGAT1 reduces oil gland size in mice and dogs, but only mice experience hair loss.

Mouse genital development depends on male or female hormones for specific features.

Removing GRK2 in skin cells causes hair loss similar to immune-related alopecia.

The improved surgical method increases success and reduces fetal loss in fetal mouse models for scarless skin healing.

Anorexia nervosa may cause a hair defect called pili torti due to malnutrition and high vitamin A levels.

Loss of Dnmt3a and Dnmt3b increases aggressive skin tumors by affecting PPAR-γ.

Wnt1a helps keep cells that can grow hair effective for potential hair loss treatments.

DAVID syndrome is a condition with immune system and hormone deficiencies, needing early diagnosis to avoid serious complications.

HLD10 can include increased body hair and Mongolian spots.

The PML protein helps prevent skin cancer in mice.

Current murine models need improvement for better human wound healing research translation.

Wnt-10b is important for keeping mouse skin cells healthy for hair growth.

Collagen XVIII is crucial for maintaining tissue structure and function in the brain, kidneys, and hair.