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CD36-expressing dermal sheath cells help form blood vessels in hair follicles, aiding hair growth.

Most U.S. keratinocyte carcinoma patients are older white males living in urban areas.

Canine claws have complex structures with different keratin types, similar to hair and nails.

New cells join the hair's dermal papilla during the growth phase, possibly affecting hair thickness.

The role of γδT-cells in causing alopecia areata remains unclear.

The hair defect is due to abnormal inner root sheath keratinization.

Cathepsin L deficiency causes hair and skin issues in mice.

Keratinocytes help keep hair follicle cells and skin cells separate in 3D cultures, which is important for hair growth research.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

Certain genes help dermal papillae cells in hair follicles grow and group together.

Skin stem cells in hair follicles are important for touch sensation.

Epidermal keratinocytes start wound healing and inflammation after schistosome infection.

Monocyte-derived dendritic cells play a key role in UVB-induced skin sensitivity and inflammation.

CD200 is not a reliable marker for identifying stem cells in all skin types.

Dermal sheath cells can help grow new hair follicles and show promise in treating hair loss.

Hair follicle stem cells help maintain skin health by moving to and supporting the skin's surface layers.

Cells that move well may improve hair loss treatments by entering hair follicles.

A unique hair tumor with a rippled pattern was identified, showing incomplete differentiation and unusual cell arrangements.

Alopecia areata involves unique activation of certain immune cells.

Keratin mutations may cause scarring alopecia by damaging hair structure.

Nonpalmoplantar skin cells can be made to express keratin 9 by interacting with palmoplantar fibroblasts.

The keratin network in mouse skin changes during cornification and affects the skin's protective barrier.

iNKT cells can help prevent and treat alopecia areata by promoting hair regrowth.

Different types of cells in the eye express specific keratins at various stages of development.

A Taiwanese patient had hair loss and skin bumps without the usual gene mutation, suggesting other genetic factors might be involved.

The woman was diagnosed with lichen sclerosus, a rare skin condition, after initial misdiagnosis and ineffective treatments.

Cathepsin E is crucial for normal skin cell differentiation and development.

The study suggests that a specific type of immune cell, memory-like NK cells, may increase during active hair loss in Alopecia areata.