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Pigs without the Hairless gene showed skin and thymus changes, useful for studying human hair disorders.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

Dimethylarsinic acid speeds up skin tumor growth in certain mice.

Mice need pantothenic acid to make inositol.

The gut microbiome affects the development of alopecia areata.

Activated β-catenin affects hair growth and skin thickness, and changes are reversible.

Multiphoton microscopy effectively images mouse skin layers and structures.

Stump-tailed macaque best for researching hair loss causes and treatments.

FoxN1 overexpression in young mice harms immune cell and skin development.

Chronological age affects the mouse auditory cortex more than hearing loss, and the brain may be less sensitive to mitochondrial dysfunction.

Iron deficiency in mothers causes hair loss in their baby mice.

Overexpression of IKZF1 and Ikaros causes hair loss in mice similar to alopecia areata.

Mutations in the PTPRQ gene cause significant balance issues in mice due to hair bundle defects in the inner ear.

EPI-NCSCs from hair follicles may help treat brain development issues in mice.

Aralia continentalis root extract may help with hair growth.

Bim and Puma proteins are found in developing mouse hair follicles and are involved in more than just cell death.

Fluorescent proteins in mouse models effectively visualize tumor blood vessel growth.

Tranexamic acid turns white hair brown in certain mice by affecting specific proteins.

A high-fat diet may weaken tongue structure by reducing certain protein genes.

Soy oil and genistein reduce hair loss risk in mice.

A gene mutation in Lama3 is linked to a common type of hair loss.

Excessive putrescine causes hair loss in transgenic mice by disrupting hair follicle development.

The naked mutation in mice causes hair loss and helps identify keratin genes.

Abnormal keratin expression in mice causes severe oral issues, affecting feeding.

Removing REDD1 in mice increases skin fat by making fat cells larger and more numerous.

Removing a key gene in mice leads to premature aging and loss of stem cells.

WWOX deficiency in mice causes skin and fat tissue problems due to disrupted cell survival signals.

Androgen receptors in the mouse brain may explain cognitive and mood changes in prostate cancer treatment.

New imaging techniques show testosterone delays hair growth and shrinks follicles in mice, but have limited depth for viewing.