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AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

A specific pathway involving AR, miR-221, and IGF-1 plays a key role in causing common hair loss.

Loss of Rap1 protein speeds up heart aging in mice.

Chemotherapy and radiation therapy cause skin and hair damage by altering gene expression and signaling pathways.

SK2 channels help control sensory signals in rat muscle spindles and hair follicles.

New therapies for Birt–Hogg–Dubé syndrome are being developed based on understanding the FLCN gene's role.

Two new gene clusters important for hair formation were found on human chromosome 11.

Increasing PBX1 reduces aging and cell death in hair follicle stem cells by boosting SIRT1 and lowering PARP1 activity.

Basal cell carcinomas need extra mutations to grow from small to large tumors.

Scientists can control how skin stem cells divide by using different treatments.

Communication between blood vessel and hair follicle cells decreases with age, affecting hair growth and blood vessel formation.

NEDD4 and NEDD4L help control intestinal stem cells and prevent tumors by breaking down the LGR5 receptor.

MIM is crucial for hair follicle formation and regeneration by controlling cilia formation and hedgehog signaling through its interaction with Cortactin and Src.

High TSPEAR levels in colorectal cancer predict worse outcomes.

KIT's role in skin cells is not entirely independent, as other cells can influence its function.

Only one of the two K16 genes on chromosome 17 makes a functional protein for keratin filaments.

Inhibiting ACE2 improves skin regeneration during tissue expansion.

BK and Kv4.2 channels help Merkel cells in rat whiskers sense touch.

Activated β-catenin affects hair growth and skin thickness, and changes are reversible.

Inhibiting TYK2 can restore hair growth in alopecia areata.

TLR3 activation helps improve skin and hair follicle healing in mice.

Tongue cancer cells show more plectin-1 than non-cancer cells, but both have similar levels of trichohyalin.

Using CDK inhibitors on rats showed a reduction in chemotherapy-caused hair loss, but later experiments could not repeat these results.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

The research found a way to develop hair growth materials by targeting a specific signaling pathway.

Cis-UCA can form new compounds without enzymes, and sulphide donors reduce its UVB-induced toxicity in skin cells.

Orai1 protein is crucial for tooth development and affects enamel thickness and mineralization.

Mice without the p21 gene can fully regenerate injured ears due to reduced Sdf1 increase and leukocyte recruitment, suggesting new ways to induce tissue regeneration in mammals.