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THH and PAD type III appear together early in hair growth, but deiminated THH appears later, suggesting another factor is involved.

Disulfide bonds are crucial for hair structure during keratinization.

The IRE gene is important for normal root hair growth in Arabidopsis plants.

MAD2B slows down the growth of skin cells that are important for hair development by interacting with TCF4.

Electrostatic interactions mainly stabilize the binding of peptides to hair keratin.

A specific mutation in PA-PLA1α causes abnormal hair growth.

QLT0267 stops hair follicle cell growth and movement.

Blocking a key energy pathway in human hair follicles can trigger stress responses that stop cell growth.

Neurosteroids help balance brain signals when certain inhibitions are reduced.

The TRPV3 channel structure changes linked to severe itch and hyperkeratosis were identified using cryo-EM.

Clim proteins are essential for maintaining healthy corneas and hair follicles.

The fuzzy gene is crucial for controlling hair growth cycles.

Keratin 19 forms less stable and shorter filaments than keratin 14, giving unique traits to certain skin cells.

Different STUB1 gene mutations cause varied symptoms in autosomal recessive ataxias.

A unique gene mutation was found in a family with monilethrix.

A new mutation, Glu402Lys, in hair keratin is linked to variable symptoms of monilethrix.

AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

JAK1 inhibitors can help reduce itchiness in atopic dermatitis.

Hard α-keratins stay stiff in water because the surrounding matrix keeps them dehydrated and strong.

XEDAR triggers a specific signaling pathway in cells.

Mouse hair follicle stem cells have a flexible chromatin state that supports skin health and hair growth.

Loss of Ten1 in mice causes telomere shortening and symptoms similar to human dyskeratosis congenita.

Activin A increases inner ear hair cell development, while follistatin decreases it.

Runx1 and CDK inhibitors help keep hair follicle stem cells inactive, affecting their future roles.

A specific gene mutation causes congenital hair loss.

Hedgehog signaling is crucial for hair development, cadherins affect cell adhesion, neutrophils play a role in skin lesions, and BP230 autoantibodies impact skin stability.