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Anti-TNF-α therapy may increase the risk of developing alopecia areata, especially in those with a history of autoimmune disease.

Alopecia areata is an autoimmune disease causing hair loss, and targeting macrophages may help treat it.

Gamma/delta T cells help defend skin against heavy metals.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

Alopecia areata patients show increased inflammation and OX40 activation, suggesting a new treatment target.

γδ T cells can prevent and treat alopecia areata, offering a new therapy option.

Dual TCR Treg cells are common in various mouse tissues and show diverse characteristics.

Alopecia areata patients have fewer protective regulatory B cells, which may contribute to the disease.

Regulatory γδ T cells help protect hair follicles from alopecia areata and promote hair regrowth.

Patients with severe active alopecia areata have lower CD200 expression and an imbalance in their immune system.

ILC1 cells contribute to hair loss in alopecia areata.

Special immune cells called Regulatory T cells help control skin inflammation and repair in various skin diseases.

Immunomodulatory therapies are effective for treating cutaneous lymphoma, particularly in early stages.

Severe alopecia areata involves higher levels of certain immune cells, which can be normalized with betamethasone.

Tofacitinib helped most patients with alopecia areata regrow hair and changes in immune cells were linked to the treatment's effectiveness.

Machine learning can predict how well patients with alopecia areata will respond to certain treatments.

IL-18 signaling helps mature Tregs move into the thymus.

Treg cells help repair and regenerate tissues by interacting with local cells.

Specific immune cells cause alopecia areata and blocking certain proteins can prevent it.

Tislelizumab can cause cutaneous lupus erythematosus.

CD3+ T-cell presence is a reliable marker to tell apart alopecia areata from pattern hair loss.

IL-4 and IL-13 might play a role in a type of hair loss similar to alopecia areata.

A specific gene change is linked to severe hair loss.

Alopecia areata involves immune system changes, especially in severe cases, with potential new treatment targets identified.

Blocking IL-12 can help treat alopecia areata by preventing hair follicle immune issues.

People with alopecia areata have fewer IL-10 producing immune cells, which might contribute to the condition.

A patient with leukemia/lymphoma also had multiple autoimmune diseases, suggesting a link between them.

New therapies aim to improve long-term hair regrowth in alopecia areata by targeting immune responses.

T cells show stress responses in alopecia areata, suggesting potential treatment targets.