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Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

Hair follicle stem cells can return to their original niche and help regenerate hair.

PDE3 inhibitor may help treat hair loss by promoting hair growth.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

Blocking mTORC1 may help prevent skin cancer by stopping the growth of certain skin stem cells.

Activating TERT in mice skin boosts hair growth by waking up hair follicle stem cells.

β-catenin in the dermal papilla is crucial for normal hair growth and repair.

Fat cells help recruit healing cells and build skin structure during wound healing.

SOX11 and SOX4 help skin cells act like embryonic cells to heal wounds in mice.

Krt15+ cells in the mouse intestine resist radiation and can start tumors.

Endoglin is crucial for proper hair growth cycles and stem cell activation in mice.

B-Raf and C-Raf are essential for maintaining melanocyte stem cells to prevent hair graying.

A hydrogel made from pig fat helps wounds heal faster by regenerating skin fat cells.

Low-dose radiation affects hair stem cell function and survival by changing their genetic material's structure.

Chrysanthemum zawadskii extract helps hair grow by stimulating hair cells.

The Bcl-2 protein is important for keeping hair follicle stem cells working and preventing hair loss.

The we/we wal/wal mice have defects in hair growth and skin layer formation, causing hair loss, useful for understanding alopecia.

The local environment is crucial for cell development in the tongue.

Disrupted stem cell signals in hairpoor mice cause hair loss.

Activated β-catenin affects hair growth and skin thickness, and changes are reversible.

Adult mouse skin contains stem cells that can create new hair, skin, and oil glands.

c-Myc activation in mouse skin increases sebaceous gland growth and affects hair follicle development.

MTS24 marks a new type of skin cell that helps hair growth and repair.

BMP and Wnt signaling balance controls hair follicle stem cell activity and hair growth.

A small group of slow-growing cells causes basal cell carcinoma to return after treatment.

Matriptase is crucial for skin barrier, hair growth, and may contribute to skin cancer.

Wnt signaling is essential for forming the skin's spinous layer through a BMP-FGF pathway.

S100A4 and S100A6 proteins may activate stem cells for hair follicle regeneration and could be potential targets for hair loss treatments.

Brg1 is crucial for hair growth and skin repair by maintaining stem cells and promoting regeneration.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.