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GCN reduces lung inflammation and damage from air pollution in mice.

Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.

Removing Sprouty genes in mice causes various hormone-related issues but does not increase cancer risk by one year of age.

Hairless USP mice have enlarged skin cysts as they age.

Genetic mutations that disrupt homocysteine breakdown lead to increased damage in mouse hair keratin.

The model using human skin on mice helps study human sebaceous glands.

ECM1-modified stem cells can effectively treat liver cirrhosis.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

Removing certain brain receptors in mice worsens seizure severity and response to treatment during hormone withdrawal.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.

Increasing COX-2 in mouse skin causes bigger sebaceous glands and thinner hair, but stopping COX-2 can reverse hair thinning.

The HCR gene contributes to psoriasis risk.

Mice with human-like EGFR had growth issues, skin defects, heart problems, and unusual bone development.

High doses of TCB cause severe health issues in marmoset monkeys.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

TCDD exposure causes sebaceous glands to differentiate and produce more lipids before shrinking.

Spiny mice have resilient, large mitochondria that help them regenerate tissue.

Nude mice with grafted human skin developed scars similar to human hypertrophic scars.

Removing the Crif1 gene in mouse skin disrupts skin balance and hair growth.

The Celsr1 gene is crucial for normal hair patterning in mice.

The G60S Connexin43 mutation causes hair growth issues and poor hair quality in mice, similar to human ODDD patients.

Activated protein C helps protect mice from radiation damage.

EPI-NCSCs from hair follicles may help treat brain development issues in mice.

A mouse model was created to study hair loss similar to humans.

The Naked (N) trait in mice is linked to lower glycine and tyrosine in hair proteins.

Removing Ctip2 in skin cells causes skin inflammation similar to atopic dermatitis.

TNF alpha in skin cells causes weight loss, hair and fat issues, and skin inflammation in mice.