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A new keratin 6 type in mice explains why some mice without certain keratin genes still have normal hair and nails.

Blocking BACE1 and BACE2 enzymes causes hair color loss in mice.

Lack of functional CYLD in mice leads to early aging and cancer.

Removing Sprouty genes in mice causes various hormone-related issues but does not increase cancer risk by one year of age.

CaBP1 and CaBP2 are important for maintaining hearing by supporting continuous calcium currents and nerve signaling in the ear.

Disrupting Bcl-xL in mice reduces skin cancer risk.

Lack of certain fatty acids causes skin, immune, and fertility issues in mice.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

Gene knockout mice developed scars similar to human hypertrophic scars, useful for studying scar progression.

Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.

Sdr16c5 and Sdr16c6 genes regulate a key point in lipid production that affects eye and skin gland function.

PCR genotyping in cre-loxP mice can be inaccurate due to unintended gene deletions in non-target tissues.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.

Certain mouse strains develop a skin condition similar to a human hair loss disease due to genetic defects.

Blocking Wnt signaling in young mice causes thymus shrinkage and cell loss, but recovery is possible when the block is removed.

B6J mice live longer before 24 months, but B6N mice live longer after; both strains show weight gain, increased food and water intake, and health issues as they age.

CXCL7 is essential for muscle repair by aiding early neutrophil infiltration.

Loss of Ten1 in mice causes telomere shortening and symptoms similar to human dyskeratosis congenita.

The PP2A-B55α protein is essential for brain and skin development in embryos.

Knocking out certain genes in mice helps understand skin and hair growth problems.

Abnormal keratin expression in mice causes severe oral issues, affecting feeding.

A missing mK6irs1 gene causes hair loss in mice.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.

The research identified new skin traits in mice, some linked to human skin conditions.

The HtrA1L364P mutation causes brain dysfunction and blood vessel damage.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

The hairless mutant gene causes early hair loss and affects skin and thymus development in mice.