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New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

Langerhans cells are crucial for skin immunity and allergic reactions.

Blocking LFA-1 prevents hair loss in mice.

Blocking the CCR5 receptor may be a new way to treat hair loss from alopecia areata.

Special cells can help regrow hair in alopecia areata.

HTLV-1-associated lichenoid dermatitis (HALD) is linked to an immune response against HTLV-1-infected cells.

Innate lymphoid cells type 1 may contribute to alopecia areata.

TGM3 is important for skin and hair structure and may help diagnose cancer.

γδT cells can protect hair follicles from alopecia areata and promote hair regrowth.

Targeting ornithine decarboxylase can help prevent skin cancer.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.

Dalpiciclib is the safest and most satisfying CDK4/6 inhibitor for advanced breast cancer patients in China.

KRTAP2-3 could help predict cancer recurrence by identifying specific cancer cells.

Anti-Desmocollin 3 antibodies can cause atypical pemphigus symptoms.

Interferon alfa-2a is effective for treating cutaneous T cell lymphoma but has significant side effects.

ILC1-like cells can cause alopecia areata by affecting hair follicles.

Lower SMAD2/3 activation predicts more severe skin cancer.

hsa_circ_0002980 can help stop liver cancer cells from growing and spreading.

TB and BCC tumors show similar follicular differentiation patterns.

CD8+ T cells re-expressing CD45RA may predict treatment resistance in severe alopecia areata.

Three molecular subtypes of advanced skin T-cell lymphoma were identified, with potential biomarkers for predicting treatment response and disease progression.

Different γδ T cell types have unique roles in causing alopecia areata.

Neutropenic patients show significant changes in immune cell types and lower neutrophil and natural killer cell percentages.

BST2 is highly expressed in certain immune cells in alopecia areata, suggesting a role in the disease.

TLR3 activation helps improve skin and hair follicle healing in mice.

Using regulatory T cells and Rapamycin together improves chronic graft-versus-host disease treatment outcomes in mice.

Rapamycin and anti-EGFR antibody reduce LAM/TSC cell migration and blood vessel growth in the uterus.

Toll-like receptor 3 helps repair the skin barrier after UV damage.