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The study found that tight junctions reach the top layer of the skin's stratum granulosum, not just the second top layer as previously thought.

Two new enzymes, PAD-R11 and PAD-R4, were cloned and showed activity, with PAD-R11 resembling epidermal enzymes.

No CD44 in alopecia areata, present in normal and androgenetic alopecia.

Giant keratoacanthoma can look like squamous cell carcinoma, requiring careful diagnosis and surgical removal.

DP and DS cells are different from DF cells in structure and function.

Immune cells boost stem cell activity in hairy moles, causing more hair growth.

The research mapped gene activity in developing mouse skin and found key markers for skin cell types and changes from fetal to early postnatal stages.

A Doberman Pinscher had a rare form of autoimmune disease causing hair loss and other severe symptoms.

Terbinafine effectively treated kerion celsi despite disrupted immune responses.

High MUC-18/MCAM levels in blood indicate a worse outlook for melanoma patients.

Melanocytes may trigger the immune response in alopecia areata, affecting hair regrowth.

Basal stem cells in the skin have distinct types that are crucial for skin structure and health.

A woman with scalp lymphoma and hair loss improved with radiotherapy, highlighting the need for biopsies in similar cases.

Trichohyalin is found in non-hair tissues and works with filaggrin in certain skin areas and conditions.

Cancer-associated fibroblasts promote tumor growth in skin cancer.

The gene LRRC15 is more active in balding areas of the scalp compared to non-balding areas.

The woman has a skin condition involving nodules, scars, and hair loss.

Hair follicle stem cells rely on nearby blood vessels for their maintenance and function.

The model successfully grew and differentiated hair follicle cells in the lab.

Keratinocyte cytokines and genetic variations influence the development of moles and skin pigmentation.

The growth of the Epstein-Barr virus in the patient's cells was linked to the worsening of her lymphoma.

Skin-resident memory T cells may contribute to chronic alopecia areata and baricitinib could be a potential treatment.

ILC1-like cells can cause alopecia areata by themselves.

p63 may influence skin cancer development and cell differentiation.

Hodgkin's lymphoma can show up as hair loss.

hHb1, hHb3, and hHb6 mRNAs start expressing at the same time in hair follicles.

BTNL2 helps protect hair follicles from immune attacks, which could aid in treating alopecia areata.