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A new keratin 6 type in mice explains why some mice without certain keratin genes still have normal hair and nails.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

The new Hairless R/J mice model improves imaging for tumor monitoring and cancer therapy evaluation.

Prolactin affects when mice shed and grow hair.

Removing REDD1 in mice increases skin fat by making fat cells larger and more numerous.

Acid inside cells speeds up aging and turns on aging signs in mice.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

Removing the Crif1 gene in mouse skin disrupts skin balance and hair growth.

The gene Foxq1, controlled by Hoxc13, is crucial for hair follicle differentiation.

Blocking EGFR in mice causes hair loss and skin changes.

A mutation in the Soat1 gene causes hair structure defects and other health issues in AKR/J mice.

Use ethical and humane practices in mouse research.

A mutation in mice causes hair loss and skin issues due to a defect in a gene affecting cell adhesion.

Mice without the Sp6 gene have problems developing several body parts, including hair, teeth, limbs, and lungs.

The study created a mouse model to better understand hair follicle stem cells' role in hair growth and repair.

The mouse model helps study and develop treatments for radiation-induced saliva reduction.

Artificially inducing hair regrowth in mice can change the normal pattern and timing of hair growth, with minimal color differences between old and new fur.

MPZL3 protein is important for controlling hair growth cycles.

The gene causing hair loss and heart issues in rough coat mice is still unknown.

MEF2C is crucial for normal hair cycle progression.

Mice with LSS deficiency showed hair loss and cataracts, similar to humans, and can help in understanding and treating this condition.

Nude mice are hairless due to a shared defect affecting both skin and thymus, not just thymic issues.

The spiny mouse is a unique menstruating rodent that can help us understand menstruation and reproductive disorders.

The N gene affects the protein makeup of mouse hair.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

PP2Acα is essential for proper hair and skin development.

Removing the p75 gene in mouse skin cells didn't affect their skin or hair growth.