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The L412F variant of TLR3 is linked to skin infections, more viral infections, and autoimmune issues.

Hair follicles help attract immune cells to the skin during stress.

The gene Ascl4 is not necessary for the development of hair, teeth, or mammary glands.

TLR3 signaling enhances the immunosuppressive properties of human periodontal ligament stem cells.

CXXC5 is a protein that prevents hair growth and could be a target for hair loss treatment.

Frizzled 3 and Frizzled 6 together control the orientation of mouse hair follicles.

A mouse model for studying scleroderma in chronic graft-versus-host disease was successfully created.

Dlx3 is essential for hair growth and regeneration.

Connexin 30 is usually absent in normal skin but can appear in certain skin conditions.

LGR5 helps maintain corneal cell characteristics and prevents unwanted changes by controlling specific cell signaling pathways.

Necl2 affects skin cell behavior and slows wound healing.

CD61 is important for mouse tooth cell growth and works through Lgr5.

CDP is crucial for lung and hair follicle cell development.

Estrogen and MLL enzymes work together to regulate genes important for hair growth and leukemia.

Lower GATA3 levels in mice help hair regrow by changing certain immune cells.

SOCS1 and SOCS3 help control skin inflammation and are important for developing treatments for skin diseases.

MEX3A is crucial for maintaining intestinal stem cells in mice.

Pannexin 3 is important for bone formation and the development of bone cells.

Cholecystokinin may help reduce skin inflammation in psoriasis.

DcR3 helps heal wounds and regrow hair by changing macrophages to a repair-focused type.

CD98hc's role in skin health decreases with age.

Ptch2 plays a key role in controlling stem cell function and the ability to regenerate after birth.

CCL 27 levels are similar in people with and without alopecia areata.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

Mice with more Flightless I protein grew back their claws better after amputation.

A specific genetic trait in tumor cells is linked to longer survival without disease in certain lymphoma patients.

Mice without the p21 gene can fully regenerate injured ears due to reduced Sdf1 increase and leukocyte recruitment, suggesting new ways to induce tissue regeneration in mammals.

The Foxn1(-/-) nude mouse shows disrupted and expanded skin stem cell areas due to high Lhx2 levels.