Search

everythinglearnresearchcommunity

for

Search:↓

Vitamin D and estrogen may help protect heart and kidney health, and maintaining sufficient vitamin D levels could be especially beneficial for African Americans, postmenopausal women, and people with chronic kidney disease.

Vitamin D receptor gene variations are not linked to alopecia areata.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.

Early detection of specific VDR mutations is crucial for effective treatment and better dental outcomes in children with hereditary vitamin D–resistant rickets.

Lack of vitamin D receptor causes hair loss in mice by allowing certain genes to overactivate.

Low vitamin D levels are common in people with Alopecia Areata.

A vitamin D receptor mutation causes rickets and affects immune responses.

Vitamin D could be important for many skin functions and synthetic versions may treat various skin diseases.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

A 4-year-old boy with a rare type of rickets and hair loss improved in bone health but not hair growth after vitamin D and calcium treatment.

1α,25-dihydroxyvitamin D3 directly affects cartilage growth and development.

Women losing hair might have lower levels of vitamin D.

Low iron levels are a significant risk factor for hair loss, while high vitamin D levels might be a response to hair loss, not a cause.

Certain variations in the vitamin D receptor gene are linked to an increased risk of polycystic ovary syndrome.

Men and premenopausal women in Korea show different patterns in iron and vitamin D levels, with no clear pattern for postmenopausal women.

Vitamin D receptor helps protect skin cells from UV damage and supports their growth.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

Three new gene mutations cause rickets and hair loss, treatable with high calcium and calcidol, but hair regrowth is rare.

Vitamin D levels are not linked to alopecia areata.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Vitamin D receptor FokI gene variation is not linked to alopecia areata.

Lysosomal proteases and cytoplasmic enzymes help hair follicle cells develop and differentiate.

Vitamin D receptor helps prevent skin tumors.

Men with early hair loss have lower vitamin D levels.

Immunofluorescence tomography is a cost-effective method for creating detailed 3-D images of tissues.

Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

Vitamin D protects skin cells from damage caused by captopril.

Vitamins A, B, C, and D are important for skin health and are being explored for new uses, but more research is needed to confirm their effectiveness.

Certain gene variations in the Vitamin D receptor may increase the risk of chronic hair loss.