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A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.

The new Hairless R/J mice model improves imaging for tumor monitoring and cancer therapy evaluation.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Anti-Desmocollin 3 antibodies can cause pemphigus-like symptoms in mice.

A new depilation method using cold wax reduces injury and improves hair growth studies in mice.

A new mutation in mice causes crooked whiskers and messy hair.

The frizzy mouse and hairless rat mutations are due to changes in the Prss8 gene.

The scraggly mutation causes hair loss and skin defects in mice.

Mice without certain skin enzymes have faster hair growth and bigger eye glands.

A new mouse mutation causes skin and hair defects due to a gene change.

Anti-Desmocollin 3 antibodies can cause atypical pemphigus symptoms.

Dsg1 is essential for maintaining a healthy skin barrier in mice.

Mice with extra human KLK14 had hair and skin problems, including weaker cell bonds and inflammation, linked to Netherton syndrome.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Mice genetically modified to produce more Del1 protein had faster hair regrowth.

The study created a mouse model to better understand hair follicle stem cells' role in hair growth and repair.

Removing the p75 gene in mouse skin cells didn't affect their skin or hair growth.

A gene mutation in mice causes increased mast cells and disorganized hair follicles in their skin.

PCR genotyping in cre-loxP mice can be inaccurate due to unintended gene deletions in non-target tissues.

A gene mutation in mice causes permanent hair loss and skin issues.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

Use ethical and humane practices in mouse research.

ODC transgenic mice can model human hair loss with skin lesions.

The PP2A-B55α protein is essential for brain and skin development in embryos.

Deleting the CDSN gene causes severe skin and hair problems, leading to death.

A gene deletion causes the "hairless" trait in Iffa Credo rats.

A gene mutation worsens skin irritation in mice due to a lack of certain fats.

Mice with the ab gene have abnormal sebaceous gland development, affecting sebum production.