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Scientists used stem cells to create a model of the skin disease Epidermolysis Bullosa simplex, which helped them understand its molecular mechanisms and could aid in finding treatments.

Dermal EZH2 controls skin cell growth and differentiation in mice.

Ceramide Synthase 4 is crucial for healthy skin barrier function.

Different keratins have unique expression patterns in mouse skin cells.

Dysplastic nevi have unique gene expressions, making them distinct from common melanocytic nevi.

KLHL24-mutant stem cells help understand skin and heart disease.

The BMP/Smads pathway and Id2 gene control hair follicle stem cells, affecting their rest and growth phases.

A new mouse mutation causes skin and hair defects due to a gene change.

SOCS1 and SOCS3 help control skin inflammation and are important for developing treatments for skin diseases.

Specific proteins and molecules play key roles in the development of human hair follicles.

Type I keratin genes are closely linked to the rex locus on mouse chromosome 11, affecting hair development.

Dermal EZH2 controls skin cell development and hair growth in mice.

The keratin 5 mutation in a family with epidermolysis bullosa simplex was due to mosaicism, not a new mutation.

Most Recessive Dystrophic Epidermolysis Bullosa patients with a specific mutation likely have Sephardic ancestry from about 500 years ago.

Acidic keratin genes are on CFA9 and basic keratin genes are on CFA27 in dogs.

Cell processes depend on time-of-day and protein complex flexibility for skin health.

Sostdc1 controls the size and number of hair and mammary gland structures.

Aging reduces dermal sheath cells, affecting youthful skin appearance.

Keratin proteins in epithelial cells are dynamic and crucial for cell processes and disease understanding.

A new genetic model may improve treatment and diagnosis for certain inherited skin diseases.

New treatments targeting specific genes show promise for treating keratin disorders.

The human keratin 6a gene's specific sequences trigger expression in skin layers after injury.

Hdac1 and Hdac2 help maintain and protect the cells that control hair growth.

Mutations in specific genes cause different types of ectodermal dysplasias.

SOX9 helps determine stem cell roles by interacting with DNA and proteins that control gene activity.

Removing SIX1 in fat cells reduces skin fibrosis.

Genetic variations affecting skin structure play a key role in severe acne.