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Blocking 5α-reductase can harm memory and brain structure, and increase harmful brain changes in male mice used for Alzheimer's disease research.

Finasteride poorly inhibits type 1 5AR, affecting its effectiveness.

2-Hydroxy-1,4-naphthoquinone is a strong 5α-reductase inhibitor.

DHT deficiency increases iNOS expression in rat testis and epididymis.

Blocking a specific enzyme in male rat fetuses leads to the development of nipples and feminized genitalia.

Finasteride reduces DHT levels and affects gene expression in fish, similar to its effects in humans.

Finasteride safely lowers DHT levels without affecting testosterone.

Daprodustat, combined with PAβN, could be a new antibacterial treatment.

Doxazosin mesylate and saw palmetto cause genetic changes in fruit flies.

The models can help find better inhibitors for conditions like baldness and prostate disorders.

Blocking the enzyme that turns testosterone into DHT can safely and effectively treat enlarged prostate.

Deferasirox effectively reduces iron overload in ß-thalassemia patients but may cause some manageable side effects.

DFMO may help control hair growth and treat cancer.

Store Tectona grandis leaf extracts in slightly acidic, light-protected conditions for best stability.

MK-386 and finasteride together effectively reduce DHT levels, potentially treating acne and male pattern baldness.

Some treatments can improve skin's defense against damage, but overuse may cause other skin problems.

Neurosteroids help control dopamine responses in the brain.

FOXN1 is crucial for thymus development and immune response in Xenopus laevis.

A new compound effectively inhibits human 5α-reductase 1.

Finasteride improves heart function in obese and aging male rats by reducing oxidative stress and p53 protein levels.

Certain amino acids in 5AR1 and 5AR2 are crucial for binding and resistance to Finasteride.

Testosterone's muscle-building effects do not require its conversion to DHT.

New tests detect finasteride and dutasteride in urine quickly and easily.

Retinol acetate can reduce the harmful effects of the drug benoxaprofen on white blood cells.

New nonsteroidal molecules can potentially increase dihydrotestosterone in neurons by blocking certain enzymes, without affecting prostate and seminal vesicle weight.

Epristeride's metabolism involves key metabolites and proteins, affecting its use in doping tests.