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A new mutation in the HR gene is linked to a rare form of hair loss with limb deformities.

pCLCA2 protein may help maintain skin structure and function.

Secreted inhibitors of Wnt and IGF signaling control hair and tooth development, creating species-specific patterns.

Too much parathyroid hormone-related protein in skin disrupts hair growth in mice.

A new gene mutation linked to KID syndrome was found, expanding genetic knowledge.

A missing mK6irs1 gene causes hair loss in mice.

Mutations in the hHb6 gene cause the hair disorder monilethrix.

Psoriasis patients' immune response to a hair protein depends on their specific gene type.

GRHL3 is important for controlling gene activity in skin cells during different stages of their development.

Targeting NMMHC IIA may help treat blood-brain barrier damage.

ICAM-1 helps regulate hair growth cycles and skin remodeling.

Fatp4 is crucial for healthy skin development and function.

Folliculin helps regulate energy and nutrient sensing, impacting Birt–Hogg–Dubé syndrome.

A protein from fat-derived stem cells, DKK1, is linked to hair loss and blocking it may help treat alopecia areata.

Keratoacanthoma comes from hair follicle cells.

A peptide from hair follicle stem cells can boost hair growth.

Truncated LTBP-1 disrupts TGF-β signaling, affecting hair growth.

Angiopoietin-1 helps hair follicle cells survive and grow, potentially treating hair loss.

CD2 might be a new treatment target for patchy alopecia areata.

Kdm6b is crucial for skin cell differentiation.

Accurate diagnosis of APL is crucial to avoid unnecessary treatments.

PAK4 is crucial in cancer progression, brain development, and could be a therapeutic target, especially through the PAK4-CREB axis.

RAPK is a rare skin disorder with pigmented spots, mainly on hands and feet, starting in youth.

A specific genetic change in the KRT71 gene causes a hair loss condition in Hereford cattle.

Keratin mutations cause skin diseases and could lead to new treatments.

Matrical tumors share a common growth mechanism involving the Wnt pathway and consistent PHLDA1 expression.