Search

everythinglearnresearchcommunity

for

Search:↓

Extracellular vesicles and androgen receptors may help identify prostate cancer resistance and reduce SARS-CoV-2 infection.

HPV16-transformed cells can change human skin cell properties, aiding tumor growth.

Activating Kras in mouse skin causes excess skin and hair loss.

Understanding how acne develops in different diseases could lead to new treatments.

Mutations in the ACTB gene cause Becker’s nevi and may lead to muscle issues in Becker’s nevus syndrome.

LGR5 is a common marker of hair follicle stem cells in different animals and is important for hair growth and regeneration.

Loss of Dnmt3a and Dnmt3b increases aggressive skin tumors by affecting PPAR-γ.

PandaOmics uses AI to find new disease treatment targets and biomarkers.

Melanoma's complexity requires personalized treatments due to key genetic mutations and tumor-initiating cells.

New therapies and trials are needed for Merkel cell carcinoma, a tough skin cancer.

Cancer risk is linked to the balance of mutations and environmental factors, not just the number of mutations.

Overexpression of HDGF in melanocytes does not cause cancer.

Cell polarity signaling controls tissue mechanics and cell fate, with complex interactions and varying pathways across species.

Chemistry42 effectively creates and optimizes new molecules for drug discovery.

IL-1 receptor absence in mice leads to skin cysts and changes in immune response after UVB exposure.

Scalp melanoma is more common and easier to diagnose early in people with androgenetic alopecia due to sun damage.

Blocking specific proteins can help remove aging cells and might treat age-related diseases and promote hair growth.

PRSS35 enzyme may help start skin tumors and could be a target for cancer treatment.

Collagen and TGF-β2 help maintain hair cell shape and youthfulness.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

The risk of skin tumors becoming malignant depends on the specific skin cell type affected.

AP-1 controls tumor cell type by affecting key signaling pathways.

The type of tumor suppressor gene lost affects the behavior of skin cancer.

UV exposure reduces Lgr6+ stem cells in mouse skin and they don't significantly contribute to skin cancer development.

Key genes linked to hair growth and cancer were identified in hairless mice.

A gene mutation in mice causes hair loss, weak bones, and protein buildup, showing how protein processing issues can lead to diseases.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Cox-2 significantly contributes to the development and progression of skin and esophageal cancers.