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The technique effectively shows how human skin and hair cells form into ball-like structures.

Endothelin-1 helps amelanotic melanocytes stick and move better on certain proteins.

Some types of extracellular matrix can change how human skin cells grow but don't affect their basic functions.

Collagenase IV helps control hair growth by affecting certain growth factors.

Multiphoton microscopy can effectively detect early endometrial cancer by analyzing collagen changes.

Amelanotic melanocytes from hair follicles are immature and likely don't transfer melanosomes to keratinocytes.

Two cases showed skin abnormalities without bone or neural defects.

Trichohyalin is found in non-hair tissues and works with filaggrin in certain skin areas and conditions.

The newborn's skin condition improved over time, leaving only lighter skin patches.

Keloid fibroblasts cause itch and pain by releasing more histamine.

Trichohyalin is also found in the outer layers of normal human skin.

Functionalized collagen scaffolds applied prenatally greatly improve skin regeneration.

Basement membrane changes are crucial for hair follicle development.

Human hair follicle cells can be grown successfully on floating collagen membranes without extra support.

Outer root sheath cells consistently express certain keratins influenced by their environment.

Collagen XVII is crucial for preventing hair and pigmentation loss by maintaining melanocyte stem cells.

The study found that tight junctions reach the top layer of the skin's stratum granulosum, not just the second top layer as previously thought.

Follicular mucinosis causes significant damage to hair follicle cells.

Skin cell clumping for hair growth is improved by a protein called fibronectin, which helps cells stick and move better.

Basement membrane supports fibroblast aggregation, aiding hair follicle development.

A rare skin condition causes red, dark, bumpy facial lesions.

A new method was developed to create better skin models for healing and reconstruction.

Certain bacteria can enhance skin regeneration.

Resident memory T cells and necroptosis may drive fibrosis in eosinophilic fasciitis and morphea.

The protein structures in the hair and tendon were preserved, but their molecular arrangements changed.