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A disease causing skin issues in young adult German short-haired pointers is hereditary, with most affected dogs not responding to treatment.

The specific skin disease variant p.(Arg2000Trp) in plectin can cause a wide range of symptoms, which should be considered when diagnosing patients.

Ezh2 controls skin development by balancing signals for dermal and epidermal growth.

Scarred skin in lichen planopilaris loses immune cells due to a decrease in a specific protein in skin cells.

The study found that certain three-dimensional scaffolds can help regenerate hair effectively.

Increasing type 17 collagen reduces aging signs in skin cells caused by UV light.

The document concludes that detailed clinical descriptions of seven family cases help understand dominant dystrophic epidermolysis bullosa's symptoms and inheritance.

Human basal cell carcinomas and squamous cell carcinomas have unique gene expression patterns not fully mirrored in mouse models.

Lymphocytes, a type of immune cell, are crucial for wound healing as they help remodel damaged areas and reduce inflammation.

Nanoplastics can penetrate skin cells, triggering inflammation and immune responses.

PD‐L1 and PD‐L2 may not effectively control immune activation in alopecia areata.

Human anagen hair follicles have unique carbohydrate patterns during keratinization.

Immune checkpoint inhibitors can cause scalp inflammation and hair follicle issues.

Skin tumor cells in patients with tuberous sclerosis have higher levels of a protein called cathepsin B.

The analyses helped identify different skin diseases in the two dogs.

NGAL indicates abnormal skin cell differentiation.

Serotype 4 of Ureaplasma showed the highest pathogenicity in female mice.

Exosomes help hair follicle development in cashmere goats.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Scientists found ways to identify and collect skin stem cells, which vary by skin area and are delicate.

The hamartoma is an abnormal hair growth with limited development in the upper hair follicle.

STIM1 is essential for sweat secretion.

Different γδ T cell types have unique roles in causing alopecia areata.

The epidermis is crucial for hair growth.

Eosinophilic infiltrate is not a reliable indicator for diagnosing chronic alopecia areata.

Matriptase is crucial for skin barrier, hair growth, and may contribute to skin cancer.

Mutations in β1 integrins cause embryonic death but have milder effects on skin.

Too much keratin 16 in mice skin causes abnormal skin thickening and structure.

Microfluidic models improve testing for aging, wound healing, and oral tissue, reducing animal testing.

Epidermal stem cells create and maintain skin structures like hair and nails through specific signaling pathways and vary by location and function.