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Alloxan diabetes, methylthiouracil, cortisone, and adrenaline affect how white mice hair follicles use glucose and cystine and their cell division.

DKK4 can be used to improve wool quality in Zhexi Angora rabbits.

A cat's poor wound healing was linked to a genetic deletion in the COL5A1 gene.

NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

K10 may not prevent tumors as previously thought and might increase benign tumor risk.

A gene variant in KRT71 causes the curly fur in Selkirk Rex cats.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.

Researchers created a new mouse model, G4, that mimics human PCOS symptoms and links the condition to a specific gene.

Dkk4 protein is necessary for the proper development and arrangement of hair follicles.

Spiny mice regenerate skin better than laboratory mice due to larger hair bulges, more stem cells, and different collagen ratios.

The method successfully isolates hair follicle stem cells from mice for research.

Overexpression of hurpin in mice leads to abnormal skin and higher skin cancer risk.

The mouse model shows potential for understanding and improving scarless wound healing, and Wnt-4 and TGF-β1 play a role in wound healing and scar formation.

Overexpression of a specific receptor in mice skin causes skin thinning, early skin barrier formation, eye issues, and hair loss.

A new method can detect mutations in mice by observing changes in hair follicle cells.

Lack of TrkC receptor delays hair follicle development.

A mutation in the hairless gene speeds up severe itchy skin in mice on a special diet.

Abnormal gene expression related to keratin causes hair loss in certain mice.

The IRF2BP2 gene affects sheep fleece quality by influencing fiber traits.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

Mouse models help understand alopecia areata and find treatments.

Mice without certain skin enzymes have faster hair growth and bigger eye glands.

ODC transgenic mice can model human hair loss with skin lesions.

SKH1 hairless mice have identifiable epidermal stem cells with specific markers.

CTHRC1 is essential for healing and preventing heart rupture after a heart attack.

The hairy ear mutation in mice is linked to changes in gene expression affecting hair growth.

The study found that variations in hair protein genes are likely due to evolutionary deletions or duplications.

A mutation in mice causes hair loss and skin issues due to a defect in a gene affecting cell adhesion.

Keratinocyte growth factor significantly alters skin and tissue development.