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Scientists found 53 keratin genes in yaks that are important for hair growth and share similarities with those in other animals.

GKLF/KLF4 and Sp1 control Keratin 19 gene activity, influencing cancer-related changes.

Inverted follicular keratosis can look like cancer but is actually a harmless tumor.

Keratinocyte gene expression is controlled by multiple modules with specific binding sites.

Glycoproteins are present in the cell membranes of various keratin fibers.

Overexpressing PKCα in mice skin increases inflammation but doesn't affect tumor growth.

Keratin biomaterials from human hair help nerve regeneration by activating Schwann cells.

SDF-1/CXCL12 and its receptor CXCR4 are crucial for melanocyte movement in mouse hair follicles.

Trichoblastomas in rabbits are linked to uncontrolled embryonic hair growth and have distinct histological features.

Stromal cells in melanoma promote tumor growth and spread.

Keratin treatment reduces astrocyte reactivity and inflammation.

GLI1 might protect against the start of skin cancer and is not linked to cancer severity.

Stem cells in mouse nails are found in the nail matrix and may control nail growth.

The surrounding tissue plays a crucial role in the growth and spread of skin cancer.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

Targeting Midkine can help reduce pain and itching in keloids.

Rabies virus was found in specific skin cells of rabid dogs' muzzles, suggesting these cells could help diagnose rabies.

A new mutation in the Connexin 26 gene was found in a patient with KID syndrome, expanding the known disorders linked to this gene.

Mcl-1 can activate Wnt signaling in skin cells, promoting growth and possibly cancer.

ZNF750 and MPZL3 are important in causing seborrheic dermatitis.

LHTric-1 is a specific antibody useful for studying hair and nail formation.

Hair follicle stem cells can become corneal-like cells with the right environment.

The human keratin 6a gene's specific sequences trigger expression in skin layers after injury.

Dysplastic nevi have unique gene expressions, making them distinct from common melanocytic nevi.

A mutation in the KRTHB5 gene causes hair and nail issues.

The nail matrix has a reduced immune response, protecting it from autoimmunity.

ERK activation spreads between cells in mouse skin, linked to cell division and influenced by TPA and EGF receptors.

Autophagy in skin cells is important for preventing inflammation, skin tumors, and controlling hair growth timing.