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Bone marrow and hair follicle cells help form skin tumors, suggesting new treatment targets.

A specific type of immune cells, called CD301b-expressing macrophages, are crucial for skin repair processes.

Neonatal blood vessels rearrange and stabilize as adults, with adult vessels better at self-repair after injury.

Collagen XVII and CD151 affect cell movement, with CD151 inhibiting migration when bound to integrins.

Lgr5+ stem cells do not cause skin tumors.

Bone marrow-derived cells contribute to skin tumors, suggesting new treatment targets for non-melanoma skin cancers.

A 3D co-culture model improved stem cell function and wound healing.

Some brain cancer cells avoid immune system detection, and certain treatments could target this to slow their growth; also, certain fat cell precursors help regenerate hair and skin after injury.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

Tiny fat-derived particles can help repair soft tissues by changing immune cell types.

Cells that move well may improve hair loss treatments by entering hair follicles.

Cell adhesion molecules are important in the development of certain skin diseases.

Skin-associated cartilage cells can influence hair growth by altering specific signaling pathways.

Scarred skin in lichen planopilaris loses immune cells due to a decrease in a specific protein in skin cells.

Lysophosphatidic acid boosts stem cell growth and movement by creating reactive oxygen species.

Proinflammatory fibroblasts and vascular endothelial cells are key in keloid development.

SLE can cause unusual gastrointestinal symptoms, and immunosuppressive therapy can be effective.

The muscle damage was caused by T-cell large granular lymphocytic leukemia, not dermatomyositis.

Special gut cells help stem cells move to and fix injured areas by activating a specific signaling pathway.

Langerhans cells and melanocytes migrate to the skin and hair follicles during early human development.

Lef1 helps stem cells become hair cells by interacting with specific signaling pathways.

Hair follicle cells produce VEGF, which can promote blood vessel cell growth and movement.

Lung and liver macrophages protect our tissues and their dysfunction can cause various diseases.

TCF/Lef1 is essential for skin barrier function by regulating lipid metabolism.

A 3D cell model can rejuvenate stem cells to improve wound healing.

Unique fat cells near fibrotic areas contribute to systemic sclerosis progression.

CD36-expressing dermal sheath cells help form blood vessels in hair follicles, aiding hair growth.

Innate lymphoid cells type 1 may contribute to alopecia areata by damaging hair follicles.

TSLP affects atopic dermatitis by increasing sebum and reducing fat through IL-4/IL-13 signaling.