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Cicatricial alopecia, a permanent hair loss condition, is mainly caused by damage to specific hair follicle stem cells and abnormal immune responses, with gene regulator PPAR-y and lipid metabolism disorders playing significant roles.

Mercury exposure damages small nerve fibers in rats' skin.

Chemical treatments and light exposure damage hair proteins.

Sunekos® effectively improves labia majora appearance and is well-tolerated.

NLRP1 helps melanoma tumors grow by boosting inflammasome activation and reducing caspase-3 activity.

Ethosomes enhance skin penetration better than liposomes, benefiting therapeutic and cosmetic applications.

Higher activity in lichen planopilaris is linked to certain immune and tissue genes.

Combining L-cysteine, NAC, and a MET inhibitor significantly kills cervical cancer cells.

Environmental factors can cause mutations in skin proteins, leading to skin disorders.

Lipases, especially gehB, are crucial for Staphylococcus aureus to enter and colonize hair follicles.

Cats infested with Lynxacarus radovskyi can lose hair without inflammation, treatable with selamectin.

Cadd4 effectively reduces cholesterol levels without side effects.

Blocking SCD1 in the skin with XEN103 shrinks sebaceous glands in mice.

Mesotherapy might help reduce fat in specific areas for those close to their ideal weight, but more research and care are needed to ensure safety.

Mesenchymal stem cells and their secretome may offer a safe and effective treatment for systemic lupus erythematosus.

SLE patients experience hair loss similar to telogen effluvium, with hair damage and immune activity at hair follicles.

Lipase H is important for hair follicle function and shaping hair fibers.

Lichen planopilaris may be an autoimmune disease causing hair loss due to immune system issues in hair follicles.

Exosomes from umbilical cord stem cells help protect hair color and health under stress.

Alopecia Areata can develop without perforin-mediated cytolysis.

A mutation in mice causes hair loss and skin issues due to a defect in a gene affecting cell adhesion.

Chronic bacterial infections of hair follicles can cause ongoing skin inflammation.

Immune cells in Hidradenitis suppurativa become more inflammatory and may be important for treatment targets.

Cow milk sugars increase fat production and inflammation in skin oil cells.

The substances improved hair regrowth and protected hair cells in humans and mice.

Allergic cats with yeast overgrowth improved with antifungal treatment.

The muscle damage was caused by T-cell large granular lymphocytic leukemia, not dermatomyositis.

The cream effectively reduces oily skin, improves hydration, and changes skin bacteria.

A woman with X-linked chronic granulomatous disease developed lupus-like skin lesions, improved with treatment, suggesting a unique skin condition in carriers.

Deleting the MAD2L1 gene is tolerated in certain mouse cancer models.